Abstract:In carbon monoxide poisoned mice enhanced glycogenolysis and lactate production in the brain are not prevented by pretreatment of the animals with reserpine or an adrenergic β-receptor blocking agent (Kö 592). Since, furthermore, glycogenolysis is not accompanied by changes in the phosphorylase a content or phosphorylase a/phosphorylase (total) ratio, it becomes unlikely that adrenergic mechanisms play a major role in the stimulation of carbohydrate breakdown during carbon monoxide poisoning. It appears more l… Show more
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