Catch-up Growth in Infants Born Small for Gestational Age - A Longitudinal Study

Amador-Licona, Norma; Martínez-Cordero, Claudia; Guízar-Mendoza, Juan Manuel; Malacara, Juan Manuel; Hernández, Javier; Alcalá, Juan F

doi:10.1515/jpem.2007.20.3.379

Cited by 17 publications

(11 citation statements)

References 21 publications

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“…A further six studies comprising 356 children reported no association between postnatal growth, measured as weight, height or BMI between birth to eight years, and insulin resistance at the ages of one to eight years, using the homeostatic model assessment for insulin resistance or fasting insulin 17, 30, 31, 37, 40, 43 (Table S1b). Positive associations were reported by eight studies with adjustments for current adiposity and four studies without.…”

Section: Resultsmentioning

confidence: 99%

Systematic review indicates postnatal growth in term infants born small‐for‐gestational‐age being associated with later neurocognitive and metabolic outcomes

et al. 2017

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We systematically reviewed papers published in English between 1994 and October 2015 on how postnatal weight gain and growth affect neurodevelopment and metabolic outcomes in term‐born small‐for‐gestational‐age (SGA) infants. Two randomised trials reported that enriched infant formulas that promoted early growth also increased fat mass, lean mass and blood pressure (BP), but had no effect on early neurocognitive outcomes. Meanwhile, 31 observational studies reported consistent positive associations between postnatal weight gain and growth with neurocognitive outcomes, adiposity, insulin resistance and BP. Conclusion: Few intervention studies exist, despite consistent positive associations between early growth and neurocognition in term‐born SGA infants.

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Section: Resultsmentioning

confidence: 99%

Systematic review indicates postnatal growth in term infants born small‐for‐gestational‐age being associated with later neurocognitive and metabolic outcomes

et al. 2017

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“…By contrast, infants who are born following IUGR exhibit significantly higher leptin values than normal children at the end of their first year of life, regardless of BMI (30). Among SGA children, those who demonstrate lower leptin levels at birth show catch‐up growth in BW and higher abdominal fat as 1‐year‐old children (31), which may relate to insulin resistance and body fat mass (22,32).…”

Section: Discussionmentioning

confidence: 99%

IGF‐I, leptin and active ghrelin levels in very low birth weight infants during the first 8 weeks of life

et al. 2009

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“…When women with PCOS become pregnant, the offspring are more likely to be exposed to elevated prenatal androgen concentrations and also to have an increased likelihood for low birth weight (260, 261). This low birth weight is often compensated for with postnatal catch-up growth, which is a risk factor for insulin insensitivity, obesity, and diabetes later in life (262), particularly in children who are not breastfed or who are weaned from the breast early (263-265). Furthermore, reduced fetal growth appears to be associated with precocious adrenarche, early puberty, poor fertility, and PCOS during later life stages (266).…”

Section: Complex Interactionsmentioning

confidence: 99%

Female reproductive disorders: the roles of endocrine-disrupting compounds and developmental timing

Crain

Janssen

Edwards

et al. 2008

Fertility and Sterility

388

233

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Objective To evaluate the possible role of endocrine-disrupting compounds (EDCs) on female reproductive disorders emphasizing developmental plasticity and the complexity of endocrine-dependent ontogeny of reproductive organs. Declining conception rates and the high incidence of female reproductive disruptions warrant evaluation of the impact of EDCs on female reproductive health. Design Publications related to the contribution of EDCs to disorders of the ovary (aneuploidy, polycystic ovary syndrome, and altered cyclicity), uterus (endometriosis, uterine fibroids, fetal growth restriction, and pregnancy loss), breast (breast cancer, reduced duration of lactation), and pubertal timing were identified, reviewed, and summarized at a workshop. Conclusion(s) The data reviewed illustrate that EDCs contribute to numerous human female reproductive disorders and emphasize the sensitivity of early life-stage exposures. Many research gaps are identified that limit full understanding of the contribution of EDCs to female reproductive problems. Moreover, there is an urgent need to reduce the incidence of these reproductive disorders, which can be addressed by correlative studies on early life exposure and adult reproductive dysfunction together with tools to assess the specific exposures and methods to block their effects. This review of the EDC literature as it relates to female health provides an important platform on which women’s health can be improved.

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Catch-up Growth in Infants Born Small for Gestational Age - A Longitudinal Study

Abstract: Female gender, early weaning, lower birth weight, and lower leptin concentration at birth are related to weight CUG in Mexican infants.

Cited by 17 publications

References 21 publications

Systematic review indicates postnatal growth in term infants born small‐for‐gestational‐age being associated with later neurocognitive and metabolic outcomes

Systematic review indicates postnatal growth in term infants born small‐for‐gestational‐age being associated with later neurocognitive and metabolic outcomes

IGF‐I, leptin and active ghrelin levels in very low birth weight infants during the first 8 weeks of life

Female reproductive disorders: the roles of endocrine-disrupting compounds and developmental timing

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