Cat-Scratch Disease

Cat-scratch Disease with Pneumonia

“…The railway journey is contingent to the sunburn but is not the cause of the sunburn. To take a strictly practical point of view, the seasidesunburn could have been avoided by shutting down the railways, and in the same way lung cancer could be largely avoided by closing the cigarette factories, quite independently of whether lung cancer and smoking have a causal or a con- Since Lee Foshay, in America, recognized cat-scratch disease as an entity (Daniels and MacMurray, 1951) excellent reviews of the condition have appeared in France (Debrd et al, 1950), in America , and in Great Britain (Brand and Finkel, 1956). In addition numerous case reports have appeared in the literature.…”

Cat-scratch Fever

“…Lange (1951) points out that the cat sharpens its claws on tree-trunks, bones, and even garbage cans, and because of its habit of frequently licking its paws, the transference of the virus from mouth to claw, or vice versa, is easily accomplished, thus explaining how the disease may follow scratch or bite. The disease has also been known to follow local lesions due to wood, thorns, bone, mosquito bites (Mollaret et al, 1951), and even inhalation of contaminated material (Daniels and MacMurray, 1951a), but a history of contact with cats is almost always found. Of 160 cases described by Daniels and MacMurray (1954), only in 12 was a history of contact with cats lacking.…”

“…The disease has also been known to follow local lesions due to wood, thorns, bone, mosquito bites (Mollaret et al, 1951), and even inhalation of contaminated material (Daniels and MacMurray, 1951a), but a history of contact with cats is almost always found. Of 160 cases described by Daniels and MacMurray (1954), only in 12 was a history of contact with cats lacking.…”

“…This tendency has dangerous potentials for the future of scientific medicine." In 1932 Lee Foshay, of Cincinnati, whilst investigating tularaemia, recognized a separate disease entity in individuals who had been scratched by cats (Daniels and MacMurray, 1951a). The condition was characterized by an initial lesion at the site of inoculation, followed by regional adenitis and fever, the adenitis progressing to a bacteriologically sterile suppuration.…”

“…The condition was characterized by an initial lesion at the site of inoculation, followed by regional adenitis and fever, the adenitis progressing to a bacteriologically sterile suppuration. In 1945 F. M. Hanger and H. Rose, of New York, developed an antigen from a similar case (Hanger himself), which gave a local reaction when injected intradermally, and was successfully tried by Foshay on his own cases (Daniels and MacMurray, 1951a).…”