Cardiogenic shock due to acute severe mitral regurgitation complicating acute myocardial infarction: a report from the SHOCK Trial Registry

Thompson, Christopher R.; Buller, Christopher E.; Sleeper, Lynn A.; Antonelli, Tracy A.; Webb, John G.; Jaber, Wael A.; Abel, James G.; Hochman, Judith S.

doi:10.1016/s0735-1097(00)00846-9

Cited by 243 publications

(156 citation statements)

References 15 publications

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“…Firstly, it is rare for cardiogenic shock to develop only a few hours after the onset of symptoms; the highest risk of shock is normally two to seven days after the myocardial infarction, when healing has begun. 5 In this particular patients. 10 However, Monin et al found that only 5% of patients required transoesophageal echocardiography to adequately quantify the severity of mitral insufficiency.…”

Section: Discussionmentioning

confidence: 73%

The Difficult Diagnosis of Ischaemic Papillary Muscle Rupture: Case report from an urban emergency department

Braun¹,

Ricklin²,

Exadaktylos³

2016

SQUMJ

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Section: Discussionmentioning

confidence: 73%

The Difficult Diagnosis of Ischaemic Papillary Muscle Rupture: Case report from an urban emergency department

Braun¹,

Ricklin²,

Exadaktylos³

2016

SQUMJ

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“…Mitral regurgitation classically courses with acute left heart failure with important pulmonary congestion 14 , which is very dissimilar from the patient's clinical evolution, being highly unlikely.…”

Section: E71mentioning

confidence: 97%

Caso 3: Mulher idosa com infarto agudo do miocárdio apresentou choque súbito durante administração de trombolítico

Bittencourt¹,

Staniak²,

Aiello³

2010

Arq. Bras. Cardiol.

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The laboratory assessment disclosed levels of urea: e68 mg/dl, creatinine: 1.2 mg/dl, glycemia: 189 mg/dl and creatine kinase: 42 U/l. A diagnosis of ongoing myocardial infarction was achieved and TNK-tPA (Tecneplase -a genetically modified form of t-PA, with increased PAI-1 resistance, increased fibrin specificity and longer half-life) ficiwas administered as bolus of 40 mg with non-fractionated heparin as bolus of 6,060 UI/kg, followed by 1,000 UI/h IV. A residual retrosternal pain persisted.One hour after the start of the treatment, the patient presented right hypochondrial pain, followed by cardiogenic shock. Volume replacement and dopamine were administered. The control electrocardiogram (April 7, 2001, 2: 17 PM) showed sinus tachycardia, HR of 110 bpm, QS complexes with ST-segment elevation in uI, III, aVF, and from V 1 to V 6 ( Figure 3). A rescue angioplasty was indicated. However, the patient presented cardiac arrest with pulseless electrical activity, did not respond to the resuscitation maneuvers and died. Clinical aspectsIn a case such as the one described here, of a 72-yearold female patient, with history of coronary artery disease and myocardial revascularization carried out more than 10 years before, who sought emergency medical assistance due to typical precordial pain, the initial evaluation must always include an electrocardiogram (ECG). A normal ECG or an ECG with non-specific alterations or signs of ischemia would lead to an initial approach that would be different from the one described here. The admission ECG of this patient showed ST-segment elevation.Considering that the patient had been submitted to a previous myocardial revascularization (MR), the correlation of the diagnostic findings of the ECG deserves special attention. First, the presence of chronic coronary artery disease, as well as the previous MR without the appropriate description of the grafts used in the surgery makes it difficult to establish the electrocardiographic correlation with the culprit artery, due both to the presence of bypasses and the possible existence of an extensive collateral network. It is also important to remember that, in spite of the native artery lesions, the probability of lesions in bypasses is of 38% 1 . Additionally, the history of chronic coronary disease in elderly diabetic women and the QS in the anterior wall raises the possibility of previous myocardial infarction in the anterior wall, and, as a consequence, the possible correlation of the ST-segment elevation corresponding with the presence of aneurysm in the same wall. Still, the existence of ST-segment elevation in A 72-year-old female patient sought medical assistance due to precordial pain that had lasted for two hours.

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“…A Surgical Case of Papillary Muscle Rupture after Acute Myocardial Infarction with Cardiogenic Shock Akira SEZAI 1, 3) , Kenji AKIYAMA 1, 3) , Seiji FUKUSHIMA 2,4) , Satoshi KASHIWAZAKI 1, 3) , Ayako TAKASAKA 1, 3) , Motomi SHIONO 3) , Yasutaka TAKAGI 2,4) , Masaaki NAGASHIMA 2,4) , Toshiyuki TANAKA 2,4) , Takaharu YAHATA 2,4) , Yasuo WATANABE 2, 4) , Rikitake KOGAWA 2,4) , Toru OSHIMA 2,4) , Atsushi HIRAYAMA 4) and Hitoshi KANAYA Papillary muscle rupture is considered to be one of the least frequently observed complications of acute myocardial infarction (AMI) and has a significantly high mortality. We report a case of a patient who survived due to an emergency surgery for papillary muscle rupture after AMI with cardiogenic shock.…”

Section: )mentioning

confidence: 99%

A Surgical Case of Papillary Muscle Rupture after Acute Myocardial Infarction with Cardiogenic Shock

明¹,

謙次²,

聖二³

et al. 2011

Nichidai Igaku Zasshi

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Cardiogenic shock due to acute severe mitral regurgitation complicating acute myocardial infarction: a report from the SHOCK Trial Registry

Abstract: The data highlight opportunities for early identification and intervention to potentially decrease the devastating mortality and morbidity of severe post-myocardial infarction MR.

Cited by 243 publications

References 15 publications

The Difficult Diagnosis of Ischaemic Papillary Muscle Rupture: Case report from an urban emergency department

The Difficult Diagnosis of Ischaemic Papillary Muscle Rupture: Case report from an urban emergency department

Caso 3: Mulher idosa com infarto agudo do miocárdio apresentou choque súbito durante administração de trombolítico

A Surgical Case of Papillary Muscle Rupture after Acute Myocardial Infarction with Cardiogenic Shock

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