Cardiac free wall rupture after acute myocardial infarction  Clinical and pathological analysis.

Yang, Xing Sheng; Sun, Jing; Huang, Da

doi:10.1536/ihj.26.935

Cited by 5 publications

(1 citation statement)

References 8 publications

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“…The cardiac rhythm commonly seen at the time of rupture includes sinus bradycardia, junctional rhythm, complete heart block, or idioventricular rhythm. 98 An echocardiogram reveals pericardial effusion and may show signs of cardiac tamponade including RV diastolic collapse and >25% respiratory variation in mitral inflow. The ventricular wall defect may also be appreciated on echocardiography.…”

Section: Clinical Presentation/diagnosismentioning

confidence: 99%

Post Myocardial Infarction Cardiogenic Shock

Yeghiazarians

2011

J Intensive Care Med

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Cardiogenic shock is often a devastating consequence of acute myocardial infarction (MI) and portends to significant mortality and morbidity. Despite improvements in expediting the time to treatment and enhancements in available medical therapy and reperfusion techniques, cardiogenic shock remains the most common cause of mortality following MI. Post-MI cardiogenic shock most commonly occurs as a consequence of severe left ventricular dysfunction. Right ventricular (RV) MI must also be considered. Mechanical complications including acute mitral regurgitation, ventricular septal rupture, and ventricular free-wall rupture can also lead to cardiogenic shock. Rapid diagnosis of cardiogenic shock and its underlying cause is pivotal to delivering definitive therapy. Intravenous vasoactive agents and mechanical support devices may temporize the patient's hemodynamic status until definitive therapy by percutaneous or surgical intervention can be performed. Despite prompt management, post-MI cardiogenic shock mortality remains high.

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Section: Clinical Presentation/diagnosismentioning

confidence: 99%