DESCRIPTIONA young man in his late 20s, presented with new-onset exertional angina. Five months earlier, the patient had presented to the trauma unit with a penetrating injury to the left chest and cardiac arrest due to cardiac tamponade. The patient had undergone emergency median sternotomy for repair of a 15 mm laceration to the right ventricular outfl ow tract. On examination, he had normal vital signs with no abnormal heart sounds or signs of heart failure. ECG was normal. Cardiac catheterisation demonstrated a communication between the proximal left anterior descending coronary artery (LAD) just before its fi rst diagonal branch and the pulmonary trunk (PT) ( fi gure 1 , upper arrow). A small blind-ended myocardial defect was also visible transiently at the same site ( fi gure 1 , lower arrow). Both defects were dynamically occluded during systole ( fi gure 1 ). The LAD-PT fi stula measured 8 mm in diameter and the myocardial defect measured 4mm on CT ( fi gure 2 ). Laboratory investigations were normal. Surgery was performed on cardiopulmonary bypass. The PT was opened exposing an 8 mm fi stulous connection with the LAD within the right ventricular outfl ow tract, distal to the septal leafl et of the pulmonary valve ( fi gure 3 ). The fi stula was closed with 4/0 prolene suture. Coronary artery fi stulas maybe congenital or acquired abnormalities. They are identifi ed in only 0.2% of routine cardiac angiographic studies 1 and can vary widely in morphologic appearance and presentation. Acquired coronary artery fi stulas are rare, but 80% are secondary to penetrating injuries. The rarity of acquired coronary fi stulas can be attributed to the high death rate (65% to 85%) of penetrating wounds involving the coronary Images in...
Traumatic left anterior descending to pulmonary trunk fi stula