Abstract:Three patients who presented with a milky fluid in the space between the posterior surface of implanted intraocular lens (IOL) and the anterior surface of the distended posterior capsular bag are described. The patients were followed and were noted to have changes in their benign course after several years. The occurrence of this milky-fluid-filled capsular bag is extremely uncommon and is difficult to perceive if the observer is not looking for it.
“…Indeed, late CBS has been described as lacteocrumenasia or liquefied aftercataract. [7][8][9] These 2 factors, ie, insignificant anterior IOL displacement and concave lens effect, can explain the hyperopic shift. A reduced myopic shift was also described by Theng et al 4 in the 2 late postoperative cases (case 7, 2 years postoperative, with a refraction of plano; case 8, 5 months postoperative, with a refraction of -0.75 diopter).…”
We report a case of late postoperative capsular block syndrome presenting with a hyperopic shift and discuss possible causes of this unusual presentation.
“…Indeed, late CBS has been described as lacteocrumenasia or liquefied aftercataract. [7][8][9] These 2 factors, ie, insignificant anterior IOL displacement and concave lens effect, can explain the hyperopic shift. A reduced myopic shift was also described by Theng et al 4 in the 2 late postoperative cases (case 7, 2 years postoperative, with a refraction of plano; case 8, 5 months postoperative, with a refraction of -0.75 diopter).…”
We report a case of late postoperative capsular block syndrome presenting with a hyperopic shift and discuss possible causes of this unusual presentation.
“…5 Rarely, postoperative lacteocrumenasia may be seen as milky-white, fluid-filled capsular bag deposits that are characterized by the accumulation of liquefied substances in a closed chamber between the posterior surface of an implanted IOL and the posterior capsule on occlusion of the anterior capsulorhexis opening. 6,7 In the present case, the pseudohypopyon was characterized by quick movement (within seconds) on positional changes. A true hypopyon is slow moving (within a few minutes) if the sediments contain low fibrin levels (ie, ocular Behçet disease); it is completely immobile if the sediments contain high fibrin concentrations (ie, HLA-B27-related uveitis).…”
This is the first reported case of a crystalline pseudohypopyon presenting as a layer of "snowdrift deposits" that settled inferiorly in the anterior chamber of the eye of a 55-year-old man and was associated with freely floating crystals in the aqueous humor, producing a "snowy Christmas Eve" appearance. The cause was spontaneous openings in the center of the anterior capsule with leaching of the crystalline substance into the anterior chamber. A dense membranous cataract was left after the lens material was partly absorbed. There was no history of trauma, surgery, or known systemic disease, and the patient presented with a unilateral, mobile, 4.0 mm white pseudohypopyon with no global pain, photophobia, or lacrimation. Although the eye was not injected on admission, repetitive head shaking resulted in dusky-red perilimbal hyperemia. A B-mode ultrasound revealed an advanced retinal detachment, and electroretinographic recordings were undetectable. Visual acuity decreased to hand motions in the affected eye, and the patient ultimately required anterior chamber paracentesis. Such sediments should be differentiated from other forms of true or pseudohypopyons because the course, treatment, and prognosis are different for each.
“…Many treatment options exist, including the use of pars plana vitrectomy and surgical or Nd:YAG laser capsulotomy in cases with a milky fluid. 1,2,4,6,7 This aggressive approach has been propagated by reports of Propionibacterium acnes in the fluid, 5 although in that case, as in the one we report, there was no anterior or vitreous chamber inflammation to suggest endophthalmitis.…”
Section: Discussionmentioning
confidence: 49%
“…When this process occurs in the late postoperative period, the captured fluid may be opacified and resemble the consistency of milk. [5][6][7][8] Several techniques to treat capsular distension syndrome have been described. The neodymium:YAG (Nd:YAG) laser can be used to create an anterior or posterior capsulotomy to provide an exit for the accumulated fluid.…”
We describe a new approach to treat late-onset capsular distension syndrome in which the fluid in the capsular bag is cloudy and prevents a posterior neodymium:YAG (Nd:YAG) laser capsulotomy. A peripheral laser iridotomy is created through which the anterior lens capsule peripheral to the IOL optic is accessed. This opening in the iris provides an access point through which an anterior Nd:YAG laser capsulotomy can be performed. Following disruption of the anterior lens capsule, the capsular fluid is released into the anterior chamber and absorbed through the inherent drainage system of the eye. This approach avoids the need for a more invasive surgical intervention.
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