1996
DOI: 10.3109/17453679608994662
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Callus formation in femur and tibia during leg lengthening:7 patients examined with DXA

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Cited by 11 publications
(7 citation statements)
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References 7 publications
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“…The animals were sacrificed by an overdose of anesthetic. The right femur and lumbar vertebrae L2–L5 were dissected for the measurements of bone mineral content and bone mineral density by dual‐energy X‐ray absorptiometry (DCS‐600; Aloka, Tokyo, Japan) according to the method of Tanaka et al (20) …”
Section: Methodsmentioning
confidence: 99%
“…The animals were sacrificed by an overdose of anesthetic. The right femur and lumbar vertebrae L2–L5 were dissected for the measurements of bone mineral content and bone mineral density by dual‐energy X‐ray absorptiometry (DCS‐600; Aloka, Tokyo, Japan) according to the method of Tanaka et al (20) …”
Section: Methodsmentioning
confidence: 99%
“…4 Although plain radiographs are the gold standard in following up distraction osteogenesis 6,15,29,38 they do not provide accurate information regarding the course of new bone formation because they are not quantitative. 3,26,33,36 The current authors decided to use DEXA, which is quantitative and has been used successfully in followup observations of distraction osteogenesis in animal models and in patients. 12,13,19,23,26,33 Observation and assessment of the regenerate is easy because the gap produced by distraction of the osteotomy is immediately definable on the images provided by DEXA.…”
Section: Discussionmentioning
confidence: 99%
“…Bone density was measured on Days 20,35,45, and 55 of the experiment, after the lengthening phase had been concluded to obtain a time-related assessment of mineralization. The most common method of analysis 19,36 was used, in which the region of interest is the area included between the osteotomy ends and the extensions of the neighboring cortices.…”
Section: Discussionmentioning
confidence: 99%
“…Хрящевые зоны этих костей могут быть гипопластичными или аномально гиперплазированными, результатом чего является резкое недоразвитие костей в длину. О биохимической природе этой формы хондродистрофий ничего не известно, если не считать сведений о различных отклонениях активности ряда ферментов, значение которых остается пока неясным [12,13].…”
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