Calcified aneurysm of the vein of Galen—successful removal

Morais, Josaphat Vilela de; Lemos, Sérgio

doi:10.1016/0090-3019(82)90131-8

Cited by 4 publications

(1 citation statement)

References 6 publications

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“…Hemodynamic factors include intracranial venous dilatation, elevated venous pressure, acute or chronic cerebral ischemia from congestive heart failure or by the steal phenomenon, or compression of the adjacent structures by the aneurysmal mass and dilated veins. Direct pressure on the brain stem can produce a comatose state, leading to a need for immediate surgical intervention [20]. …”

Section: Symptoms/presentationmentioning

confidence: 99%

Spontaneous Thrombosis of Vein of Galen Aneurysmal Malformation

1999

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Two cases of aneurysmal malformations of the vein of Galen (AVG) with spontaneous thrombosis are reported. Angiogram and MRI before thrombosis demonstrated AVGs with slow arteriovenous shunts and associated stagnation of contrast in the venous sac secondary to severe outflow restriction. Based on these findings, one patient was managed conservatively, and the other underwent placement of a ventriculoperitoneal shunt. Surveillance of the lesions with subsequent MRIs revealed spontaneous thrombosis of the AVGs with excellent clinical outcomes. Proposed mechanisms of spontaneous thrombosis include slow flow shunts, obstruction of the venous outflow or obstruction of the feeding artery. Similar cases in the literature are reviewed with special emphasis on diagnostic tests, symptomatology, mechanisms of thrombosis and therapeutic options.

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Section: Symptoms/presentationmentioning

confidence: 99%

Spontaneous Thrombosis of Vein of Galen Aneurysmal Malformation

1999

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Hydrodynamics in vein of Galen malformations

et al. 1992

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Forty-three patients with vein of Galen aneurysmal malformations (VGAM) referred to us for endovascular treatment between 1985 and 1990 and 335 additional cases published in the literature were reviewed with particular attention to the presence of ventricular enlargement and outcome after shunting. Hydrocephalus was the second most frequent symptom (46.8%); it is more frequent in infants (73%) than in children, adults (30%) or neonates (15%). Of the patients reported in the literature, 17.9% had undergone shunting. Within the shunted population there was an overall morbidity of 41% and a mortality of 10% (especially in the infant group). In our series 17 patients (39.5%) were shunted and a significant difference in the clinical outcome was noted between the shunted and the nonshunted group. Of the nonshunted patients, 66.6% were free of any neurological deficit or mental retardation and fewer than 5% presented with significant mental retardation. On the other hand, only 33.3% of the shunted patients had a favorable outcome and more than 15% developed significant mental retardation. Among the various causes of hydrocephalus in patients with VGAMs, such as obstruction of the aqueduct, subarachnoid hemorrhage, or ex vacuo hydrocephalus, high venous pressure may be of particular importance. In this article a physiopathological interpretation of the hydrodynamics in VGAMs is developed and a speculative explanation for CSF disorders related to ventricular shunting proposed. Treatment of hydrocephalus in VGAMs can be achieved through obliteration of the malformation or at least diminishing the venous pressure; surgical ventricular shunting does not have to be the first treatment of hydrodynamic disorders associated with VGAMs, especially in infants.

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Vein of Galen aneurysm and hydrocephalus

Rocco

1991

Child's Nerv Syst

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Calcified aneurysm of the vein of Galen—successful removal

Cited by 4 publications

References 6 publications

Spontaneous Thrombosis of Vein of Galen Aneurysmal Malformation

Spontaneous Thrombosis of Vein of Galen Aneurysmal Malformation

Hydrodynamics in vein of Galen malformations

Vein of Galen aneurysm and hydrocephalus

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