C-reactive protein and particulate matter predict plasma fibrinogen levels

Su, Ta-Chen; Wu, Ching-Fen; Chan, Chang-Chuan; Chen, Ming-Fong

doi:10.1016/j.ijcard.2011.09.019

Cited by 4 publications

(2 citation statements)

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“…The mechanisms linking air pollution and CVD are proposed in the scientific statement from the American Heart Association by Brook et al (2010) , and our previous studies may add support for these mechanisms ( Chen et al 2014 ; Chuang et al 2007 ; Wu et al 2010 , 2012 ). Seasonal variation in PM 10 levels predicted seasonal variations in plasma fibrinogen levels in a panel study of middle-aged adults ( Su et al 2011b ). In addition, this study provides supporting evidence of an increased risk of atherosclerosis following long-term air pollution exposure in Asia.…”

Section: Discussionmentioning

confidence: 99%

Carotid Intima-Media Thickness and Long-Term Exposure to Traffic-Related Air Pollution in Middle-Aged Residents of Taiwan: A Cross-Sectional Study

Hwang

Shen

et al. 2015

Environ Health Perspect

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BackgroundAssociations between long-term exposure to air pollution and carotid intima-media thickness (CIMT) have inconsistent findings.ObjectivesIn this study we aimed to evaluate association between 1-year average exposure to traffic-related air pollution and CIMT in middle-aged adults in Asia.MethodsCIMT was measured in Taipei, Taiwan, between 2009 and 2011 in 689 volunteers 35–65 years of age who were recruited as the control subjects of an acute coronary heart disease cohort study. We applied land-use regression models developed by the European Study of Cohorts for Air Pollution Effects (ESCAPE) to estimate each subject’s 1-year average exposure to traffic-related air pollutants with particulate matter diameters ≤ 10 μm (PM10) and ≤ 2.5 μm (PM2.5) and the absorbance levels of PM2.5 (PM2.5abs), nitrogen dioxide (NO2), and nitrogen oxides (NOx) in the urban environment.ResultsOne-year average air pollution exposures were 44.21 ± 4.19 μg/m3 for PM10, 27.34 ± 5.12 μg/m3 for PM2.5, and (1.97 ± 0.36) × 10–5/m for PM2.5abs. Multivariate regression analyses showed average percentage increases in maximum left CIMT of 4.23% (95% CI: 0.32, 8.13) per 1.0 × 10–5/m increase in PM2.5abs; 3.72% (95% CI: 0.32, 7.11) per 10-μg/m3 increase in PM10; 2.81% (95% CI: 0.32, 5.31) per 20-μg/m3 increase in NO2; and 0.74% (95% CI: 0.08, 1.41) per 10-μg/m3 increase in NOx. The associations were not evident for right CIMT, and PM2.5 mass concentration was not associated with the outcomes.ConclusionsLong-term exposures to traffic-related air pollution of PM2.5abs, PM10, NO2, and NOx were positively associated with subclinical atherosclerosis in middle-aged adults.CitationSu TC, Hwang JJ, Shen YC, Chan CC. 2015. Carotid intima–media thickness and long-term exposure to traffic-related air pollution in middle-aged residents of Taiwan: a cross-sectional study. Environ Health Perspect 123:773–778; http://dx.doi.org/10.1289/ehp.1408553

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Section: Discussionmentioning

confidence: 99%

Carotid Intima-Media Thickness and Long-Term Exposure to Traffic-Related Air Pollution in Middle-Aged Residents of Taiwan: A Cross-Sectional Study

Hwang

Shen

et al. 2015

Environ Health Perspect

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“…Seasonal variation of cardiovascular events may be linked to the changes of cardiovascular and endocrine/metabolic markers in different seasons [34]. However, the carotid IMT demonstrated a long-term surrogate outcome of subclinical atherosclerosis, which provides clear evidence of potential cardiovascular beneficial effects of forest environments.…”

Section: Discussionmentioning

confidence: 99%

The Health Effects of a Forest Environment on Subclinical Cardiovascular Disease and Heath-Related Quality of Life

et al. 2014

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BackgroundAssessment of health effects of a forest environment is an important emerging area of public health and environmental sciences.PurposeTo demonstrate the long-term health effects of living in a forest environment on subclinical cardiovascular diseases (CVDs) and health-related quality of life (HRQOL) compared with that in an urban environment.Materials and MethodsThis study included the detailed health examination and questionnaire assessment of 107 forest staff members (FSM) and 114 urban staff members (USM) to investigate the long-term health effects of a forest environment. Air quality monitoring between the forest and urban environments was compared. In addition, work-related factors and HRQOL were evaluated.ResultsLevels of total cholesterol, low-density lipoprotein cholesterol, and fasting glucose in the USM group were significantly higher than those in the FSM group. Furthermore, a significantly higher intima-media thickness of the internal carotid artery was found in the USM group compared with that in the FSM group. Concentrations of air pollutants, such as NO, NO2, NOx, SO2, CO, PM2.5, and PM10 in the forest environment were significantly lower compared with those in the outdoor urban environment. Working hours were longer in the FSM group; however, the work stress evaluation as assessed by the job content questionnaire revealed no significant differences between FSM and USM. HRQOL evaluated by the World Health Organization Quality of Life-BREF questionnaire showed FSM had better HRQOL scores in the physical health domain.ConclusionsThis study provides evidence of the potential beneficial effects of forest environments on CVDs and HRQOL.

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Urban climate and cardiovascular health: Focused on seasonal variation of urban temperature, relative humidity, and PM2.5 air pollution

Tsao

Hwang

Chen

et al. 2023

Ecotoxicology and Environmental Safety

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C-reactive protein and particulate matter predict plasma fibrinogen levels

Cited by 4 publications

References 11 publications

Carotid Intima-Media Thickness and Long-Term Exposure to Traffic-Related Air Pollution in Middle-Aged Residents of Taiwan: A Cross-Sectional Study

Carotid Intima-Media Thickness and Long-Term Exposure to Traffic-Related Air Pollution in Middle-Aged Residents of Taiwan: A Cross-Sectional Study

The Health Effects of a Forest Environment on Subclinical Cardiovascular Disease and Heath-Related Quality of Life

Urban climate and cardiovascular health: Focused on seasonal variation of urban temperature, relative humidity, and PM2.5 air pollution

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