Bullous pemphigoid-like seabies

Parodi, Aurora; Saino, M.; Rebora, Alfredo

doi:10.1111/j.1365-2230.1993.tb02197.x

Cited by 14 publications

(12 citation statements)

References 5 publications

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“…Light microscopy and direct immunofluorescence of a blister taken from our patient revealed a subepidermal bulla and IgG and C3 deposition on the dermoepidermal junction, these results were consistent with bullous pemphigoid. There are 13 previously reported cases, 3,[7][8][9][10][11][12][13][14][15][16] in eight of the 11 cases in which direct immunofluorescent study was performed, the results were positive. 9,11,12,[14][15][16] Therefore, light microscopy and direct immunofluorescent study are not sufficient to distinguish between true bullous pemphigoid and scabies with bullous lesions.…”

Section: Discussionmentioning

confidence: 99%

A case of crusted scabies with a bullous pemphigoid‐like eruption and nail involvement

Nakamura¹,

Taniguchi²,

Ohtaki³

2006

The Journal of Dermatology

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We report a case of a 71-year-old man infected at a nursing home who developed a bullous pemphigoid-like eruption with nail involvement. He was diagnosed by his family doctor as suffering from eczema and was treated with topical corticosteroids, then blisters started appearing. He was next diagnosed as suffering from bullous pemphigoid and treated with oral prednisolone, which worsened his condition. He was finally diagnosed as having crusted scabies with bullous pemphigoid-like eruptions and nail involvement at our clinic. He was then prescribed oral ivermectin (two doses of 12 mg ivermectin with a 1-week interval) and topical lindane (1%gamma-BHC in petrolatum) for scabies with 5% salicylic acid in plastibase as an additional treatment for the crusted lesions on his soles. He showed remarkable improvement in 2 weeks, and his nails showed complete recovery after 7 weeks of occlusive dressing treatment with 1%gamma-BHC. One and a half years later, the patient showed no sign of a recurrence of scabies. The histology of a blister taken from this patient was similar to that of bullous pemphigoid. Direct immunofluorescence showed immunoglobulin (Ig)G and C3 deposition at the dermoepidermal junction similar to that of bullous pemphigoid, but indirect immunofluorescence was negative. The bullous symptoms of this patient were considered to be due to the scabies, because the patient recovered completely after receiving treatment for scabies. Indirect immunofluorescent study is important to distinguish between scabies with blister formation and true bullous pemphigoid.

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Section: Discussionmentioning

confidence: 99%

A case of crusted scabies with a bullous pemphigoid‐like eruption and nail involvement

Nakamura¹,

Taniguchi²,

Ohtaki³

2006

The Journal of Dermatology

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“…[5][6][7][8] Thus, our case is the first to be acknowledged as attributable to severe phimosis. It is likely that the constricting phimotic ring may have been responsible for significant lymph stasis and the subsequent dilatation of the superficial lymphatic channels.…”

Section: (C3) At the Dermo-epidermal Junctionmentioning

confidence: 67%

“…In the literature, linear immune deposits were found in eight of 31 cases of bullous scabies. [3][4][5][6][7][8][9][10] Bullous scabies can resemble autoimmune bullous diseases clinically, by light microscopy, and immunopathologically. 1 Several cases of bullous scabies have been reported as initially misdiagnosed and treated as bullous pemphigoid.…”

Section: Editormentioning

confidence: 99%

“…The age of onset ranges from puberty to 45 years; therefore the present patient represents the most belated instance ever described. [3][4][5][6][7][8] Acquired lymphangioma presents as asymptomatic, painful, burning or pruritic, thin-walled vesicles filled with a clear fluid. However, sometimes smooth and/or hyperkeratotic papulovesicles, papules, and/or nodules may occur, often leading to misdiagnoses such as herpetic infections, molluscum contagiosum and warts.…”

Section: (C3) At the Dermo-epidermal Junctionmentioning

confidence: 99%

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A case of bullous scabies misdiagnosed as acquired epidermolysis bullosa

2015

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“…Histopathology of the blisters reveals subepidermal bullae and mites are not seen in most of them. Direct immunofluorescence is either negative (two cases) 1,2 or shows linear or granular deposits of IgG, IgM, and complement C3 in various combinations at the dermo‐epidermal junction (DEJ) 1,3–5 . Despite clinical and histological similarity, circulating anti‐basement membrane zone antibodies have been found in only one patient.…”

mentioning

confidence: 99%