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Goldberg, H.

doi:10.1103/physrevd.24.1991

Cited by 10 publications

(10 citation statements)

References 32 publications

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“…The S2 flow was first identified in high resolution K I and Na I absorption spectra by Goldberg et al (1975) and subsequently re-observed multiple times over the next couple of years (Goldberg 1979). It is interesting to compare these and Bernat et al's (1979) CO line-ofsight absorption velocities with those from the CARMA emission spectra obtained at similar spectral resolutions and also to measure, the perhaps co-spatial line broadening of the K I S2 absorption feature.…”

Section: K I 7699å Spectramentioning

confidence: 79%

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CARMA CO(J= 2 – 1) OBSERVATIONS OF THE CIRCUMSTELLAR ENVELOPE OF BETELGEUSE

O’Gorman

Harper

Brown

et al. 2012

The Astronomical Journal

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We report radio interferometric observations of the 12 C 16 O 1.3 mm J = 2 − 1 emission line in the circumstellar envelope of the M supergiant α Ori and have detected and separated both the S1 and S2 flow components for the first time. Observations were made with the Combined Array for Research in Millimeter-wave Astronomy (CARMA) interferometer in the C, D, and E antenna configurations. We obtain good u − v coverage (5-280 kλ) by combining data from all three configurations allowing us to trace spatial scales as small as 0 ′′ .9 over a 32 ′′ field of view. The high spectral and spatial resolution C configuration line profile shows that the inner S1 flow has slightly asymmetric outflow velocities ranging from −9.0 km s −1 to +10.6 km s −1 with respect to the stellar rest frame. We find little evidence for the outer S2 flow in this configuration because the majority of this emission has been spatially-filtered (resolved out) by the array. We also report a SOFIA-GREAT CO(J = 12 − 11) emission line profile which we associate with this inner higher excitation S1 flow. The outer S2 flow appears in the D and E configuration maps and its outflow velocity is found to be in good agreement with high resolution optical spectroscopy of K I obtained at the McDonald Observatory. We image both S1 and S2 in the multi-configuration maps and see a gradual change in the angular size of the emission in the high absolute velocity maps. We assign an outer radius of 4 ′′ to S1 and propose that S2 extends beyond CARMA's field of view (32 ′′ at 1.3 mm) out to a radius of 17 ′′ which is larger than recent single-dish observations have indicated. When azimuthally averaged, the intensity fall-off for both flows is found to be proportional to R −1 , where R is the projected radius, indicating optically thin winds with ρ ∝ R −2 .

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Section: K I 7699å Spectramentioning

confidence: 79%

“…high spectral resolution atomic Na and K absorption profiles (Goldberg et al 1975). 12 C 16 O was subsequently detected at 230 GHz in the J = 2 − 1 rotational emission line by Knapp et al (1980), although a search for SiO(J = 2 − 1) by Lambert & Vanden Bout (1978) had been unsuccessful.…”

Section: Introductionmentioning

confidence: 99%

CARMA CO(J= 2 – 1) OBSERVATIONS OF THE CIRCUMSTELLAR ENVELOPE OF BETELGEUSE

O’Gorman

Harper

Brown

et al. 2012

The Astronomical Journal

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“…The renal vasodilation and increase in renal blood flow produced by dopexamine HCI were blocked by SCH 23390, a potent and highly selective DA1receptor antagonist (Goldberg et a1 1984;Hilditch et all984) and this agrees with our earlier finding using bulbocapnine as a DAl-receptor blocker (Brown et al 1985b). The vascular DAl-receptor has a highly regional disposition and is particularly abundant in the renal vasculature (Goldberg 1972).…”

Section: Discussionmentioning

confidence: 99%

The cardiovascular actions of dopexamine hydrochloride, an agonist at dopamine receptors and β2-adrenoceptors in the dog

Smith

Hall

Farmer

et al. 1987

Journal of Pharmacy and Pharmacology

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The receptor pharmacology of the cardiovascular effects of dopexamine hydrochloride in the anaesthetized dog (given by i.v. infusion of 3 X 10(-9)-10(-7) mol kg-1 min-1) has been analysed by the use of selective receptor antagonists and of ganglionic blockade. The increases in cardiac output, contractility, and rate were antagonized by the beta 2-adrenoceptor antagonist, ICI 118551. Renal blood flow rose secondary to reduction in renal vascular resistance and this was antagonized by SCH 23390, a highly selective DA1-receptor antagonist. Peripheral vasodilation and reduction of blood pressure were mediated by a combination of DA1- and DA2-receptor and beta 2-adrenoceptor stimulation. In a separate group of dogs, the cardiac stimulant effects of dopexamine HCl were partially reflex and were reduced by ganglion block, revealing responses due to stimulation of cardiac beta 2-adrenoceptors. Thus the beta 2-adrenoceptor agonist action of dopexamine HCl is not only partly responsible for afterload reduction but also leads to direct cardiac stimulation. From its cardiovascular profile, dopexamine HCl is likely to be of use in acute treatment of heart failure.

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“…A t the present time, however, the existence of dopamine-containing peripheral autonomic neurons is uncertain. O n the other hand there exist differences between noradrenaline and dopamine which support the view that dopamine might in fact have some function at the periphery other than being solely a precursor substance for the biosynthesis of noradrenaline (for reviews see: HORNYKIEWICZ, 1971 ;GOLDBERG, 1972). We therefore studied the effect of dopamine on ruminal smooth muscle preparations in vitro and additionally investigated the influence of dopamine infusion on the extrinsic movements of the reticulorumen in conscious goats.…”

Section: Introductionmentioning

confidence: 93%

“…The changes in heart rate observed in our experiments with dopamine are difficult to interpret, since the cardiovascular effects can be caused by many factors changing cardiac function and peripheral resistance. In the dog, cardiovascular and renal effects of dopamine result from actions on a-and /?-adrenergic receptors and on specific dopamine receptors in the renal and mesenteric vascular beds (GOLDBERG, 1972). A small dose of dopamine stimulates cardiac contractility and output but also decreases total peripheral resistance by renal and mesenteric vasodilation.…”

Section: ; Titchen and Newhook 1968)mentioning

confidence: 99%

The Effect of Dopamine on Gastric Adrenergic Receptors in the Goat

Miert

Vugt

2010

Zentralblatt Für Veterinärmedizin Reihe A

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Summary From in vitro experiments it is concluded that there is no evidence for the existence of specific dopaminergic receptors in the ruminal smooth muscle. The responses produced by dopamine in isolated ruminal smooth muscle preparation of the goat appear to involve primarily a direct mechanism of action because inhibition of dopamine‐β‐hydroxylase failed to modify the action of this amine. Further, only large doses of cocaine caused reduction — a small one — in the response to dopamine. This mechanism of action is related to α‐adrenergic receptors because dibenamine and phentolamine were very effective antagonists in contrast to propranolol. Dopamine was about 50 to 100 times less potent than adrenaline, while adrenaline appeared to be about 1.5 times as active as noradrenaline. It is concluded that the α‐adrenergic receptors mediating the biphasic or motor responses to dopamine, noradrenaline and adrenaline are located on smooth muscle cells rather than on a nervous pathway. In the conscious goat dopamine, noradrenaline and adrenaline caused inhibition of the extrinsic reticulo‐ruminal contractions. The mean doses of noradrenaline and dopamine which were equally active to 1 μg per kg. per min. of adrenaline were approximately 2 μg and 10 μg per kg. per min., respectively. The cardiovascular effects produced by dopamine were different from those produced by adrenaline or noradrenaline. Zusammenfassung Die Wirkung von Dopamin auf die adrenergischen Magenrezeptoren der Ziege In vitro‐Versuche sprechen für das Fehlen von spezifischen dopaminergischen Rezeptoren in der glatten Pansenmuskulatur. Die Reaktionen von isolierten Präparaten von glatter Pansenmuskulatur der Ziege dürften auf einem direkten Wirkungsmechanismus beruhen, da eine Hemmung der Dopamin‐β‐Hydroxylase den Effekt dieses Amins nicht veränderte. Nur große Kokaindosen vermögen den Dopamineffekt ein wenig zu vermindern. Dieser Wirkungsmechanismus steht mit α‐adrenergischen Rezeptoren in Beziehung, denn Dibenamin und Phentolamin waren im Gegensatz zu Propranolol sehr starke Antagonisten. Dopamin war etwa 50 bis 100 mal weniger wirksam als Adrenalin, während Adrenalin ungefähr 1,5 mal aktiver war als Noradrenalin. Die α‐adrenergischen Rezeptoren, welche den biphasischen oder motorischen Effekt auf Dopamin, Noradrenalin und Adrenalin vermitteln, dürften sich eher an den glatten Muskelzellen denn an den Nerven befinden. An der wachen Ziege hemmten Dopamin, Noradrenalin und Adrenalin die Hauben‐Pausenmotorik. Die mittleren Noradrenalin‐ und Dopamindosen, welche einen gleich starken Effekt wie 1 μg Adrenalin pro kg Körpergewicht und pro Minute auslösten, betrugen 2 μg bzw. 10μg pro kg und Minute. Die kardiovaskulären Effekte von Dopamin unterschieden sich von denjenigen des Adrenalins und Noradrenalins. Résumé Action de la dopamine sur les récepteurs adrénergiques stomacaux chez la chèvre Des recherches in vitro ont montré l'absence de récepteurs dopaminergiques spécifiques dans la musculature lisse de la panse. Les réactions de préparations isolées de mus...

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Bounds one+e−→l*l

Cited by 10 publications

References 32 publications

CARMA CO(J= 2 – 1) OBSERVATIONS OF THE CIRCUMSTELLAR ENVELOPE OF BETELGEUSE

CARMA CO(J= 2 – 1) OBSERVATIONS OF THE CIRCUMSTELLAR ENVELOPE OF BETELGEUSE

The cardiovascular actions of dopexamine hydrochloride, an agonist at dopamine receptors and β2-adrenoceptors in the dog

The Effect of Dopamine on Gastric Adrenergic Receptors in the Goat

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