Blood Pressure and Heart Rate Responses in Carotid Angiography with Sodium Acetrizoate (Triurol): An Experimental Study in Cats

Lindgren, P.; Tornell, G.

doi:10.1177/028418515805000120

Cited by 3 publications

(3 citation statements)

References 5 publications

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“…Bearing in mind that these effects on the systemic circulation during carotid angiography (11) are probably attributable to a direct action of the contrast medium on nervous structures, it is interesting to consider the present results in relation to what is known about the effects of the various contrast media on the vascular permeability and the bloodbrain barrier. If the permeability is greatly affected, there may be increased passage of contrast substance through the vessel walls.…”

Section: Discussionmentioning

confidence: 95%

Carotid angiography with tri-iodobenzoic acid derivatives

Lindgren

1959

Acta Radiologica

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Section: Discussionmentioning

confidence: 95%

Carotid angiography with tri-iodobenzoic acid derivatives

Lindgren

1959

Acta Radiologica

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“…Cardiovascular reactions to injection of roentgen contrast media into the carotid artery have been studied by LINDGREN & TORNELL (1958). They measured the blood pressure and the pulse rate at injections of sodium acetrizoate into the carotid artery in cats.…”

Section: Discussionmentioning

confidence: 99%

Neurotoxicity of Roentgen Contrast Media

Jeppsson¹,

Olin²

1970

Acta Radiologica. Diagnosis

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“…Rather, a striking disturbance in vasomotion, characterized by the circulatory events summarized above (42), occurred in the absence of demonstrable vasospasm. There is an abundant literature arguing against (6,22,23,35,36) the role of vasospasm in the production of contrast medium injury. One paper presents evidence that vasospasm exerts a protective effect (13).…”

Section: Discussionmentioning

confidence: 99%

Vasopressor Potentiation of Neurotoxicity in Experimental Aortography

Margolis¹,

Yerasimides²

1966

Acta Radiologica. Diagnosis

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In 1956 MARGOLIS and associates introduced a new experimental model (39,60) for the study of the pathogenesis of the injury to the nervous system produced by contrast media used in diagnostic angiology. Employing retrograde abdominal aortography in the dog, and using the spinal cord as the site of study they described, for the first time in experimental animals, the full evolution of the contrast medium lesion. From these observations it was manifest that existing concepts of the injury as a transient vascular disorder were no longer adequate and had to be revised to include a severe necrotizing effect.Continuing their studies with this model (41, 42), they described in association with the injury a severe irreversible disruption of the circulation of the cord, immediate in onset, and characterized by an abrupt arrest of the toxic injection mass, an immediate impairment of vascular permeability barriers, torpid blood flow with development of severe sludging, and a persistent elevation of vascular resistance in the area of injury. The changes took place in the absence of demonstrable vasospasm. These disturbances in vasomotion were so severe as to constitute, in their opinion, a formidable obstacle to any therapeutic 388 at Harvard Libraries on June 30, 2015 acr.sagepub.com Downloaded from

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Blood Pressure and Heart Rate Responses in Carotid Angiography with Sodium Acetrizoate (Triurol): An Experimental Study in Cats

Cited by 3 publications

References 5 publications

Carotid angiography with tri-iodobenzoic acid derivatives

Carotid angiography with tri-iodobenzoic acid derivatives

Neurotoxicity of Roentgen Contrast Media

Vasopressor Potentiation of Neurotoxicity in Experimental Aortography

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