Biomonitoring of BTEX in primary school children exposed to hookah smoke

Tabatabaei, Zeynab; Hoseini, Mohammad; Fararouei, Mohammad; Shamsedini, Narges; Baghapour, Mohammad Ali

doi:10.1007/s11356-022-19882-9

Cited by 4 publications

(4 citation statements)

References 70 publications

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“…In order to quantify the PAH metabolites, 3 mL of this supernatant was injected into the GC–MS apparatus (Agilent, HP5-MS capillary column, 7890, USA) 22 , 23 . To adjust the concentrations of PAH metabolites, the creatinine (ng/g cr) was determined according to Jaffe’s spectrophotometric technique at the ultraviolet (500 nm) absorption spectrum 4 .…”

Section: Methodsmentioning

confidence: 99%

“…Furthermore, the HI i was computed to assess the cumulative risk of exposure to dietary PAHs (Eq. 3 ) 4 , 24 : …”

Section: Methodsmentioning

confidence: 99%

“…People may be exposed to these pollutants from different sources and routes 1 . The most common routes to get exposure to PAHs are inhalation, smoking, food, soil, air, and skin contact 4 .…”

Section: Introductionmentioning

confidence: 99%

“…Endocrine system disruption is caused by PAHs, which are toxic, cancer-causing, and mutagenic substances. The International Agency for Research on Cancer (IARC) has classified benzo[a]pyrene (BaP) as a carcinogen to humans (group 1) 4 . The US Environmental Protection Agency (USEPA) has likewise classified them as priority pollutants.…”

Section: Introductionmentioning

confidence: 99%

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Non-carcinogenic and cumulative risk assessment of exposure of kitchen workers in restaurants and local residents in the vicinity of polycyclic aromatic hydrocarbons

Shamsedini

Dehghani

Samaei

et al. 2023

Sci Rep

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Polycyclic aromatic hydrocarbons (PAHs) are often formed when organic substances do not burn completely. This study evaluates the non-carcinogenic and cumulative risks associated with PAHs levels by testing blood and urine samples in kitchen workers and residents near restaurants in Shiraz, Iran. Metabolites of PAH in the urine samples as well as clinical parameters in the blood samples were measured. The non-carcinogenic and cumulative risk assessments from exposure of the study groups to PAH metabolites were also evaluated. The highest average concentrations of PAH metabolites were related to kitchen workers (2126.7 ng/g creatinine (ng/g cr)). The metabolites of 1-Hydroxypyrene (1-OHP) and 9-Phenanthrene (9-OHPhe) had the highest and lowest mean concentrations, respectively. A direct correlation was observed between the levels of PAH metabolites with malondialdehyde (MDA) and total antioxidation capacity (TAC) levels (p < 0.05). Hazard Index (HIi) was obtained less than one (HIi < 1), indicating low-risk negative health impacts on the target groups. Nevertheless, conducting more studies to determine the health status of these people is quite evident.

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Section: Methodsmentioning

confidence: 99%

“…Furthermore, the HI i was computed to assess the cumulative risk of exposure to dietary PAHs (Eq. 3 ) 4 , 24 : …”

Section: Methodsmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 99%

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