“…Ovarian vein syndrome was first reported by Clark in 1964 1 , who attributed the rare ureteral obstruction, to the normal anatomic relationship between the ureter and the ovarian vein. Since this initial description, various authors have implicated different factors which may cause or contribute to the its pathogenesis: an increase in pressure of the dilated ovarian vein 2,3 , presence of an aberrant ovarian vein 4 , the existence of a fibrous sheath 1 , an ovarian vein thrombosis 5 , or tumors invading the vena cava. 2,6 Anatomically, the gonadal vein passes superiorly and medially to enter the inferior vena cava or renal vein and causes minimal oblique indentation with medial deviation of the ureter in the L3-L5 region and same indentation is seen as it encounters the iliac arteries at the S1 level, as reported by Dure-Smith 4 .…”