Bilateral Congenital Arteriovenous Communications (Aneurysm) of the Cerebral Vessels

Jaeger, Rudolph J.; Forbes, Roy P.

doi:10.1001/archneurpsyc.1946.02300170035005

Cited by 58 publications

(28 citation statements)

References 5 publications

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“…However, he did not seem to recognize the pathological relationship between the aneurysmal malformation and shunting. Jaeger and colleagues 15,16 were the first to describe vein of Galen "aneurysms" in 1937, a misnomer according to the current understanding of this lesion. An understanding of the true nature of these malformations has evolved over the years, from initially being thought of as a diffuse group of aneurysms and AVMs toward an understanding of their true nature as AVFs.…”

Section: ©Aans 2013mentioning

confidence: 99%

Vein of Galen aneurysmal malformations: critical analysis of the literature with proposal of a new classification system

Mortazavi

Griessenauer

Foreman

et al. 2013

PED

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Vein of Galen aneurysmal malformations are a rare and diverse group of entities with a complex anatomy, pathophysiology, and serious clinical sequelae. Due to their complexity, there is no uniform treatment paradigm. Furthermore, treatment itself entails the risk of serious complication. Offering the best treatment option is dependent on an understanding of the aberrant anatomy and pathophysiology of these entities, and tailored therapy is recommended. Herein, the authors review the current concepts related to vein of Galen aneurysmal malformations and suggest a new classification system excluding mesodiencephalic plexiform intrinsic arteriovenous malformations from this group of malformations.

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Section: ©Aans 2013mentioning

confidence: 99%

Vein of Galen aneurysmal malformations: critical analysis of the literature with proposal of a new classification system

Mortazavi

Griessenauer

Foreman

et al. 2013

PED

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“…Aneurysm of vein of galen is a dilatation either due to pressure overload or associated arterio-venous malformation. First case of vein of galen aneurysm was described by jagger et al 3 Vein of galen malformation has been classified variedly by different author. According to the Lasjaunias classification which is based on the origin and number of feeding arteries into choroidal and mural type.…”

Section: Discussionmentioning

confidence: 99%

Vein of Galen Aneurysm Diagnosed by Antenatal Ultrasonography : A Rare Case

Netam¹,

Kumar²,

Singh³

et al. 2015

jemds

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Vein of galen aneurysm is a rare congenital vascular malformation in which there is shunting of arterial flow in to dilated cerebral vein along the tectum which drains in to straight sinus. Majority of cases of perinatal mortality in case of vein of galen are caused by congestive cardiac failure. Vein of galen is classified according to clinical presentation. Due to advancement in ultrasonography and color Doppler this malformation can be detected early and management can be planed. Advancement in endovascular neurointerventional technique made possible to treat this malformation if the neonate sustained for some time. Here we present a case of vein of galen aneurysm diagnosed in a routine antenatal ultrasonography.

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“…Thus, there was no genuinely angiomatous element in the anomaly and this seems to have been true also of the other cases which are discussed here, The extent of the venous anomalies described has varied considerably and may depend in part on the size of the arteriovenous fistulae. In the case of Jaeger and Forbes (1946), in which both arteries and veins were considerably enlarged, the fistula was big enough to lead to cardiac enlargement. The fistulae and the resultant aneurysm constitute a single entity, but it is to be expected that the lesion might occur from time to time in combination with arteriovenous malformations in other parts of the brain.…”

Section: Discussionmentioning

confidence: 98%

Congenital Arteriovenous Fistula With an Aneurysm of the Great Cerebral Vein and Hydrocephalus Treated Surgically

Gibson¹,

Taylor²,

Richardson³

1959

Journal of Neurology, Neurosurgery & Psychiatry

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Aneurysms occasionally develop from the great cerebral vein as a result of congenital arteriovenous fistulae. In a recent review Hirano and Terry (1958) referred to 18 cases, nine of which were confirmed at necropsy. The example of this rare condition reported here presented as a case of hydrocephalus showing certain noteworthy features. The condition responded well to surgical treatment, but the patient died after a fall. The connexions of the aneurysm were defined at necropsy. Case ReportA baby girl of 6 months was brought for neurosurgical consultation because of obvious hydrocephalus in February, 1954. The pregnancy and delivery and the early development of the infant had been normal, and five weeks before she was examined she had been able to sit up almost unaided. At the time her balance had become impaired and she no longer raised her head spontaneously. The skull was seen to be expanded and superficial scalp veins were prominent. The skull circumference was 515 cm.; the fontanelle was half closed but tense. Eye movements and pupillary reactions were normal; no other abnormalities were detected. In radiographs of the skull, opening of sutures and finger markings of the vault were seen but there was no evidence of the cause of the increased pressure. The transverse sinuses were in their normal position and the posterior fossa was not unduly shallow. At ventriculography, the intracranial pressure was high and an obstruction of the aqueduct was demonstrated, but not with the flaskshaped outline usually found in aqueduct stenosis. The obstruction (Fig. I) began 2 cm. below the third ventricle and increased caudally giving rise to a "rat tail" shadow. The aqueduct was displaced forwards and downwards suggesting a space-occupying lesion behind the pineal. To define this further, air was subsequently introduced by the lumbar route after the ventricular pressure had been lowered. The radiological appearances then were surprising (Fig. 2). All the subarachnoid cisterns were greatly dilated, particularly the cistema ambiens. The sulci of the cerebral convexities were considerably enlarged up to the midline. These findings were interpreted to indicate defective absorption of fluid at the sagittal sinus as the primary cause of the hydrocephalus. 224It was thought that the resultant rise in fluid pressure in the subarachnoid space had caused dilatation of the cisterns to such a degree that the cistema ambiens had obstructed the aqueduct and so produced a type of hydrocephalus which was at least intermittently noncommunicating. To reverse this sequence an anastomosis between the spinal theca and the peritoneal cavity was made with polythene tubing on February 25, 1954. The immediate result was highly satisfactory: skull expansion ceased, the scalp veins became less distended, and normal development was resumed. At the age of I year the child could stand with support; the skull circumference had increased by only 2 cm. At 18 months she could stand unaided; the fontanelles were closed. She had a normal vocabulary and t...

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Bilateral Congenital Arteriovenous Communications (Aneurysm) of the Cerebral Vessels

Cited by 58 publications

References 5 publications

Vein of Galen aneurysmal malformations: critical analysis of the literature with proposal of a new classification system

Vein of Galen aneurysmal malformations: critical analysis of the literature with proposal of a new classification system

Vein of Galen Aneurysm Diagnosed by Antenatal Ultrasonography : A Rare Case

Congenital Arteriovenous Fistula With an Aneurysm of the Great Cerebral Vein and Hydrocephalus Treated Surgically

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