Benign swelling of submandibular glands under general anesthesia “anesthesia mumps”

Jafra, Anudeep; Arora, Saurabh; Dwivedi, Deepak

doi:10.1016/j.jclinane.2016.05.016

Cited by 6 publications

(3 citation statements)

References 7 publications

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“…However, postoperative acute parotitis was first encountered in 1960 ( 3 ). The acute, transient, noninfectious parotid or submandibular gland swelling associated with general anaesthesia has been defined as anaesthesia mumps ( 4 , 5 ). The possible mechanism in postoperative acute parotitis cases after general anaesthesia is thought to be obstruction of the parotid duct as a result of gland enlargement.…”

Section: Discussionmentioning

confidence: 99%

Postoperative Acute Parotitis After Cesarean Delivery Under Spinal Anaesthesia

Günaydın¹,

Bağcaz²,

Inan³

et al. 2021

Turk J Anaesthesiol Reanim

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Section: Discussionmentioning

confidence: 99%

Postoperative Acute Parotitis After Cesarean Delivery Under Spinal Anaesthesia

Günaydın¹,

Bağcaz²,

Inan³

et al. 2021

Turk J Anaesthesiol Reanim

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“…General anesthesia has long been identified as a rare cause of acute parotitis ("anesthesia mumps"). This entity is typically bilateral (14,15), but has also been reported as a unilateral occurrence (16).…”

Section: Discussionmentioning

confidence: 99%

Development of acute parotitis after non-invasive ventilation

Martínez

Dicpinigaitis

2017

J. Thorac. Dis.

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A 90-year-old woman underwent laparoscopic exploratory laparotomy for evaluation of suspected mesenteric ischemia. She was promptly extubated postoperatively and transferred to the intensive care unit, where on the first postoperative day she developed hypoxemia necessitating initiation of noninvasive ventilation (NIV) with bilevel positive airway pressure (BiPAP). After 8 hours of BiPAP, she was noted to have swelling, erythema and tenderness in the right preauricular area. Ultrasound evaluation demonstrated an enlarged right parotid gland. With discontinuation of BiPAP and supportive measures, parotitis resolved within 6 days. The mechanism of NIV-induced acute parotitis likely involves transmission of positive pressure to the oral cavity, causing obstruction to salivary flow within the parotid (Stensen) duct. Conditions that increase salivary viscosity and promote salivary stasis, such as advanced age, dehydration, and absence of salivary gland stimulation due to restriction of oral intake, may render patients more susceptible to this complication. As NIV will continue to be a commonly-used modality for the treatment of acute respiratory failure, clinicians should be aware of this phenomenon.

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“…Weight management and continuous positive airway pressure (CPAP) therapy are the primary treatments for obstructive sleep apnea syndrome and generally result in good response when properly utilized. Perioperative continuous positive pressure ventilator‐assist devices used after surgery have caused benign, self‐limited “anesthesia mumps,” and have also been implicated in dangerous intracranial insufflation of air resulting in pneumocephalus after sinus surgery . Another case involved bilateral parotitis in a patient treated with short‐term CPAP in the intensive care unit for an exacerbation of chronic obstructive pulmonary disease .…”

Section: Introductionmentioning

confidence: 99%