Bacterial Infections

Winn, Washington C.; Chandler, Francis W.

doi:10.1007/978-1-4757-3935-0_9

Cited by 10 publications

(48 citation statements)

References 523 publications

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“…Infected animals recover from the clinical illness and detectable bacteraemia occurs in only a small proportion of cases. The gross pathology of the model was consistent with bronchopneumonia [28]. Although lobar pneumonia observed in other mouse models is the usually recognised pattern of disease in young adults, atypical forms are increasingly recognised.…”

Section: Discussionsupporting

confidence: 54%

Genetic background affects susceptibility in nonfatal pneumococcal bronchopneumonia

Preston

Beagley²,

Gibson³

et al. 2004

Eur Respir J

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Section: Discussionsupporting

confidence: 54%

Genetic background affects susceptibility in nonfatal pneumococcal bronchopneumonia

Preston

Beagley²,

Gibson³

et al. 2004

Eur Respir J

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“…aeruginosa gains access to pulmonary endothelium through the distal airway following disruption of alveolar epithelium (15) or, alternatively, through the circulation following systemic infection (16). This bacterium displays a vascular tropism, especially for the lung microcirculation (32,58). However, there is no evidence for direct interaction between P. aeruginosa and pulmonary artery endothelium, since bacilli associated with the bronchovascular bundle tend to accumulate in the perivascular interstitium (56a, 13, 57).…”

Section: Discussionmentioning

confidence: 99%

Heterogeneity of pulmonary endothelial cyclic nucleotide response toPseudomonas aeruginosaExoY infection

Morrow

Seifert

Kaever

et al. 2015

American Journal of Physiology-Lung Cellular and Molecular Phys

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“…In some cases, however, stimulation of fibroblasts by the inflammatory response brings in proliferation of connective tissue (12). The resulting intraalveolar and interstitial organization and fibrosis produce organizing pneumonia.…”

Section: Discussionmentioning

confidence: 99%

Focal Organizing Pneumonia: CT and Pathologic Findings

et al. 2001

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The purpose of this study was to describe the CT findings of focal organizing pneumonia and to compare the findings with pathology. CT findings of histologically proven focal organizing pneumonias in 26 consecutive patients were analyzed. In 17 patients who had undergone surgical resections, the findings were correlated with pathology. Focal organizing pneumonias appeared as a nodule (n= 13) or a mass (n=13), ranging from 9 mm to 66 mm in diameter. Ground-glass opacity was seen in 6/13 (46%) nodules and 6.5/13 (50%) masses (k=.48) with an extent ranging from 5% to 75% (mean, 16%). In 4/26 (15%) patients, the extent was more than 50% of the lesion. They showed smooth (n=4), lobulated (n=8), spiculated (n=1), or lobulated and spiculated margin (n=13). On correlative analysis, nodule or mass on CT consisted histologically of intraalveolar exudate or microabscess, chronic inflammatory cell infiltration, fibrotic nodules, and polypoid granulation tissue in the alveolar or bronchiolar spaces. Ground-glass opacity consisted of interstitial fibrosis and chronic inflammatory cell infiltration and intraalveolar polypoid granulation tissue. Focal organizing pneumonia may simulate a lung cancer with variable appearances on CT and the findings reflect underlying histopathology of the disease.

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Bacterial Infections

Cited by 10 publications

References 523 publications

Genetic background affects susceptibility in nonfatal pneumococcal bronchopneumonia

Genetic background affects susceptibility in nonfatal pneumococcal bronchopneumonia

Heterogeneity of pulmonary endothelial cyclic nucleotide response toPseudomonas aeruginosaExoY infection

Focal Organizing Pneumonia: CT and Pathologic Findings

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