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                     <i>Mapping Anesthesia Genes</i> </b>

Cm, Crowder

doi:10.1097/00000542-199802000-00004

Cited by 6 publications

(1 citation statement)

References 12 publications

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“…[For methodological overview, the reader is referred to a recent excellent review of this technique (7).] Chromosomal regions have been identified that control sensitivity to propofol (8) and various responses to alcohol (9)(10)(11)(12).…”

Section: Introductionmentioning

confidence: 99%

Alcohol and anesthetic mechanisms in genetically engineered mice

Homanics

Quinlan²,

Mihalek³

et al. 1998

Front Biosci

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Introduction 3. Neurotransmitter receptors 3.1. Gamma-aminobutyric acid type A receptors (GABA A-Rs) 3.1.1. Gene knockout of the beta 3 subunit of the GABA A-R 3.1.2. Gene knockout of the gamma 2 subunit of the GABA A-R 3.1.3. Gene knockout of the long splice variant of the gamma 2 subunit of the GABA A-R 3.1.4. Gene knockout of the alpha 6 subunit of the GABA A-R 3.2. Serotonin receptors 3.2.1. Gene knockout of the serotonin 5-HT 1B receptor 3.3. Opiate receptors 3.3.1 Gene knockout of the opiate mu receptor 3.4. Alpha adrenergic receptors 3.4.1. Point mutation of the alpha-2a adrenergic receptor 3.4.2. Gene knockout of the alpha-2b adrenergic receptor 3.4.3. Gene knockout and overexpression of the alpha-2c adrenergic receptor 3.5. Dopamine receptors 3.5.1. Gene knockout of the dopamine D1 receptor 3.5.2. Gene knockout of the dopamine D2 receptor 3.5.3. Gene knockout of the dopamine D3 receptor 3.5.4. Gene knockout of the dopamine D4 receptor 4. Kinases 4.1. Gene knockout of fyn tyrosine kinase 4.2. Gene knockout of the gamma isoform of protein kinase C 5. Neurotransmitters 5.1. Nitric oxide 5.1.1 Gene knockout of neuronal nitric oxide synthase 6. Perspectives 7. Acknowledgements 8. References

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