“…When exposed to a perceived stressor stimuli, HPA axis is activated as consequence of a sympathetic activation, which produces increases in the heart rate, blood pressure, glucose levels, bronchodilation as well as over-activation of cognitive and metabolic resources [3][4][5]. This response has been found either in applied military combat, in where there is a life perceived risk, thus an acute stressful stimuli [6], showing increases in lactate, heart rate, blood oxygen saturation, muscular strength, salivary cortisol and blood glucose levels [7], and in clinical evaluations in undergraduate students [8], clinical stays or prior to their first written exams in the course [9][10][11][12][13].Previous studies showed how acute stress produces a reduction in blood flow in the prefrontal cortex, which means a decrease in oxygen and nutrients, triggering a decrease of the effectiveness of the executive functions associated [3,14], affecting brain regions as the hippocampus, leading to concentration difficulties and acutely reducing memory capability [15] either working and operative memory [15][16][17][18], decision making [16] and contributing to irritability, anxiety, loss of self-confidence and worriedness [19,20].…”