Attempt to quantitate relation between cardiac function and infarct size in acute myocardial infarction.

Mathey, D.; Biefield, W; Hanrath, Peter; Effert, S

doi:10.1136/hrt.36.3.271

Cited by 128 publications

(23 citation statements)

References 13 publications

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“…Despite this multiplicity of factors contributing to ventricular performance after a myocardial infarct, studies in man have suggested a gross relationship between infarct size and ventricular performance. Mathey et al (1974) divided patients with acute myocardial infarctions into three groups based on pulmonary artery diastolic pressure. They reported a direct relationship between filling pressure and enzymatically derived determinations of infarct size.…”

Section: Discussionmentioning

confidence: 99%

Myocardial infarct size and ventricular function in rats.

Pfeffer¹,

Pfeffer²,

Fishbein³

et al. 1979

Circulation Research

1,156

758

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Section: Discussionmentioning

confidence: 99%

Myocardial infarct size and ventricular function in rats.

Pfeffer¹,

Pfeffer²,

Fishbein³

et al. 1979

Circulation Research

1,156

758

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“…PESP was the most potent intervention and produced a significant augmentation in NET in group 2 crystals at 1, 2, 4, 6, 8, and 24 h after coronary occlusion but only at 1 and 2 h in NET in group 3 crystals. INTRODUCTION The extent of a myocardial infarction is an important predictor of subsequent morbidity, mortality, and residual left ventricular function in patients (1)(2)(3)(4). Current evidence suggests that a zone of jeopardized myocardium coexists with a region of central infarction (5) and that infarct extension into the jeopardized zone may be relatively common in patients (4)(5)(6)(7).…”

mentioning

confidence: 99%

Functional characterization of left ventricular segmental responses during the initial 24 h and 1 wk after experimental canine myocardial infarction.

Roan¹,

Scales²,

Saffer³

et al. 1979

J. Clin. Invest.

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“…1 2 Therefore, it might be important to estimate the volume ofjeopardized myocardium before myocardial infarction occurs.…”

mentioning

confidence: 99%

Regional myocardial volume perfused by the coronary artery branch: estimation in vivo.

Koiwa¹,

Bahn²,

Ritman³

1986

Circulation

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Seven closed-chest dogs were anesthetized with intramuscular Innovar and a N20/02 gas mixture. Maximal coronary vasodilatation was induced by intra-aortic injection of nitroglycerin (200 ,ug/kg) 157-163, 1986. IT HAS BEEN reported that extent of depression of cardiac function in patients with myocardial infarction relates to the amount of myocardium that is infarcted. 1 2 Therefore, it might be important to estimate the volume ofjeopardized myocardium before myocardial infarction occurs.The primary aim of this study was to evaluate how accurately estimates in vivo of regional myocardial volume perfused by a selected coronary artery could be made. For this purpose we used the multiple x-ray tube, high-speed computed tomographic scanner known as the dynamic spatial reconstructor (DSR).3 The secondary aim of this study was to establish whether or not there was a predictable relationship between the regional myocardial volume perfused and the cross-sectional area of the maximally dilated coronary arterial branch that perfuses the volume. For these studies we used closed-chest anesthetized dogs. Methods Preliminary study to establish drug infusion regimen to achieve maximum dilatation of epicardial and intramyocardial coronary arteries. Maximum dilatation of the coronary circulation was desired so as to reduce variability of vessel cross sections due to uncontrolled vasospasm and to enhance the delivery of radiopaque medium to the myocardium.Mongrel dogs were anesthetized with Innovar (fentanyl and droperidol) and a 2:1 N20/02 gas mixture and the airway of each was intubated for controlled respiration with an artificial respirator (Harvard App. model 607

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Attempt to quantitate relation between cardiac function and infarct size in acute myocardial infarction.

Cited by 128 publications

References 13 publications

Myocardial infarct size and ventricular function in rats.

Myocardial infarct size and ventricular function in rats.

Functional characterization of left ventricular segmental responses during the initial 24 h and 1 wk after experimental canine myocardial infarction.

Regional myocardial volume perfused by the coronary artery branch: estimation in vivo.

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