Assignment&lt;footref rid="foot01"&gt;&lt;sup&gt;1&lt;/sup&gt;&lt;/footref&gt; of the Schwannomin-Interacting Protein 1 (SCHIP1) gene to human chromosome band 3q25 by in situ hybridization and with somatic cell hybrids

Goutebroze, Laurence; Sarkissian, H. Der; Brault, Estelle; Thomas, Gilles

doi:10.1159/000048795

Cytogenet Genome Res

2001

DOI: 10.1159/000048795

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Assignment<footref rid="foot01"><sup>1</sup></footref> of the Schwannomin-Interacting Protein 1 (SCHIP1) gene to human chromosome band 3q25 by in situ hybridization and with somatic cell hybrids

Laurence Goutebroze

H. Der Sarkissian²,

Estelle Brault³

et al.

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2002

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References 6 publications

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Mouse Models of Neurofibromatosis 1 and 2

Gutmann

Giovannini

2002

Neoplasia

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The neurofibromatoses represent two of the most common inherited tumor predisposition syndromes affecting the nervous system. Individuals with neurofibromatosis 1 (NF1) are prone to the development of astrocytomas and peripheral nerve sheath tumors whereas those affected with neurofibromatosis 2 (NF2) develop schwannomas and meningiomas. The development of traditional homozygous knockout mice has provided insights into the roles of the NF1 and NF2 genes during development and in differentiation, but has been less instructive regarding the contribution of NF1 and NF2 dysfunction to the pathogenesis of specific benign and malignant tumors. Recent progress employing novel mouse targeting strategies has begun to illuminate the roles of the NF1 and NF2 gene products in the molecular pathogenesis of NF-associated tumors.

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Mouse Models of Neurofibromatosis 1 and 2

Gutmann

Giovannini

2002

Neoplasia

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Assignment<footref rid="foot01"><sup>1</sup></footref> of the Schwannomin-Interacting Protein 1 (SCHIP1) gene to human chromosome band 3q25 by in situ hybridization and with somatic cell hybrids

Cited by 1 publication

References 6 publications

Mouse Models of Neurofibromatosis 1 and 2

Mouse Models of Neurofibromatosis 1 and 2

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