Assessment of ventricular late potentials in HIV positive patients based on the RR interval histogram

Benchimol-Barbosa, Paulo Roberto; Barbosa-Filho, José Maria; De-Sa, C.A.M.; Nadal, Jurandir

doi:10.1109/cic.1999.825973

Cited by 3 publications

(2 citation statements)

References 6 publications

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“…Considering the presence of ventricular late potentials after transmural myocardial infarction, the mechanisms triggering repetitive arrhythmia are not necessarily related to the presence of these signals 11 . We speculate that other factors, such as unidirectional block 11 and an instantaneous heart rate 24 , associated with alterations in the pattern of sympathovagal autonomic myocardial modulation may contribute to the transformation of the substrate. In group B patients with documented episodes of sustained monomorphic ventricular tachycardia, all variables of the vector magnitude analyzed had durations and voltages significantly lower and more prolonged than those in group A patients for all cut-off bands, except for the DUR variable at 25Hz.…”

Section: Discussionmentioning

confidence: 99%

“…The correlation between the lesion and ventricular late potentials depends on clinical factors related to the disease, on local electrophysiological factors (ischemia), and on the type of processing applied to the acquired electrocardiographic signals. In this context, we highlight the use of drugs that spare the myocardium in the acute phase 15,22 , the start of depolarization of the affected site in relation to the total duration of ventricular activation, the type of filter used to analyze the events 19,23 , and the instantaneous heart rate 4,24 . Features like the low ability to characterize the infarct site based on the isolated analysis of surface electrocardiography should be considered at the time of group division, because it may lead to inconsistent results 10 .…”

Section: Discussionmentioning

confidence: 99%

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Analysis of the Prevalence of Ventricular Late Potentials in the Late Phase of Myocardial Infarction Based on the Site of Infarction

Paulo

Benchimol-Barbosa

Sousa

et al. 2002

Arq. Bras. Cardiol.

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Objective -The initial site of myocardial infarction (MI) may influence the prevalence of ventricular late potentials (VLP), high-frequency signals, due to the time course of ventricular activation. The prevalence of VLP in a period of more than 2 years after acute MI was assessed focusing on the initially injured wall . Methods - [1][2][3] . Due to the low velocity of conduction, these potentials exceed the duration of ventricular activation and are detected in the ST-segment. The use of statistical techniques, in which 2 to 3 hundred sequential QRS complexes detected on the body surface are independently coherent-averaged, allows amplification and identification of these signals because of the reduction in the intensity of the instrumentation noises 4-7 .The identification of ventricular late potentials in patients with a transmural myocardial infarction is of major interest 3,[8][9][10] . The myocardial remodeling process that follows transmural myocardial infarction is characterized by fibrosis, redistribution of fibers in the injured region, and residual metabolic alterations not only plays a major role in segmentary myocardial function but also provides a favorable environment for the development of reentry circuits 11 .Considering these markers, preliminary studies suggest that transmural myocardial infarctions involving the inferiorinferodorsal wall have a higher prevalence of ventricular late potentials than those involving the anterior-anteroseptal wall. These findings are based on differences related to the start of electrical activation in the affected regions 12,13 . However, studies relating the prevalence of ventricular late potentials after transmural myocardial infarction with the affected wall show some conflicting results. Some studies have focused attention on the occurrence of ventricular late potentials in the first 12 months after transmural myocardial infarction when the myocardial remodeling process is still underway 14 . Data on the prevalence of ventricular late potentials in a late phase (>2 years) of transmural myocardial infarction are very scarce. In this phase, the prevalence of ventricular late potentials reflects not only the natural

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Section: Discussionmentioning

confidence: 99%