Aspectos Nutricionales en La Enfermedad De Graves: Comunicación De Un Caso

Abstract

INTRODUCTION Graves' disease (GD) is the most common cause of hyperthyroidism and the most common autoimmune disorder in the United States (1). This disease is a common autoimmune cause of hyperthyroidism in which the thyroid gland undergoes lymphocyte infiltration, with an elevation in circulating T lymphocytes and the occurrence of autoantibodies that bind to the TSH receptor (TRAb), with consequent stimulation of gland growth and function (2). Graves' disease (GD) is characterized by diffuse goiter, thyrotoxicosis, and some organ-specific manifestations including Graves' ophthalmopathy and Graves' dermopathy. Although the production of thyroid-stimulating hormone (TSH) receptor antibodies is thought to be a crucial underlying etiology for this immunological process, the exact etiology of GD is still unknown (3).