Artificial Ventilation in Hyaline Membrane Disease

Martin-Bouyer, G; Monset-Couchard, Michelle; Bomsel, F; Larroche, Jeanne‐Claudie; C, Amiel-Tison; Minkowski, A.

doi:10.1159/000240273

Cited by 10 publications

(1 citation statement)

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“…Clinical data were collected from patients' medical records and included mother's age, presence of premature rupture of membranes (PROM), histological chorioamnionitis, antenatal glucocorticoid use, ritodrine hydrochloride use, magnesium sulfate use, gestational week at birth, birth weight, sex, RDS (chest radiograph consistent with Bomsel criteria, grades 2-4) [15], Apgar score at 1 and 5 min, delivery by caesarean section (C-section), surfactant treatment, whether the child was small for gestational age (SGA, birth weight <10th percentile or median birth weight at same gestational age in Japanese newborns) [16], and ventilator treatment.…”

Section: Methodsmentioning

confidence: 99%

Very Low Birth Weight Monochorionic Diamniotic Twins as a Risk Factor for Symptomatic Patent Ductus Arteriosus

Yamaguchi

Wada²,

Nagasawa³

et al. 2016

Neonatology

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Background: Some prior studies have shown that symptomatic patent ductus arteriosus (sPDA) is highly familial. Although it is estimated that both genetic and environmental factors may contribute to sPDA, evidence is still lacking. Objective: The aim of this study was to determine the risk factors for sPDA, focusing on the genetic and in utero environment by analyzing very low birth weight (VLBW) singletons and twins. Methods: This retrospective case-control study reviewed the medical records of 445 VLBW infants (25 weeks ≤ gestational age <32 weeks, 600 g ≤ birth weight <1,500 g) and compared the incidence of sPDA among monochorionic diamniotic (MD) twins (n = 65), dichorionic diamniotic (DD) twins (n = 66), and singletons (n = 314). Results: Stepwise multiple regression analysis showed that twin siblings (p = 0.001), gestational week (p < 0.001), antenatal steroid use (p = 0.021), and premature rupture of membranes (p = 0.002) were independent predictors of sPDA. Incidence of sPDA in MD twin siblings was significantly higher than that in singletons (p < 0.01), whereas no significant difference was found between singletons and DD twins or between MD and DD twins. Conclusions: The current results show that being a VLBW MD twin is an independent risk factor for sPDA, and that both genetic and in utero environmental factors may contribute to its development.

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Section: Methodsmentioning

confidence: 99%