Arteriovenous Malformations: A Diagnostic and Therapeutic Challenge

Miguel, Ramón de; López‐Gutiérrez, Juan Carlos; Boixeda, P.

doi:10.1016/j.adengl.2013.04.026

Cited by 14 publications

(15 citation statements)

References 65 publications

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“…Stage III is destructive form with skin changes like ulceration, bleeding and continuous pain, possibly even with necrosis and lytic bone changes. Stage IV is decompensated heart failure 9. Our patient was stage III acroangiodermatitis.…”

Section: Discussionmentioning

confidence: 75%

Parkes Weber syndrome presenting as Stewart-Bluefarb acroangiodermatitis

et al. 2019

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Parkes Weber syndrome (PWS) is a rare disorder characterised by arteriovenous (AV) fistula, along with capillary, lymphatic, venous malformations and limb hypertrophy. Stewart-Bluefarb syndrome is a variant of acroangiodermatitis, which is associated with congenital AV malformation/fistulas. It usually begins early in life, unilaterally over lower extremities presenting as violaceous to dusky coloured macules, papules or plaques with tendency to ulcerate. We are reporting a case of AV malformation fulfilling the triad of PWS and presenting with acroangiodermatitis.

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Section: Discussionmentioning

confidence: 75%

Parkes Weber syndrome presenting as Stewart-Bluefarb acroangiodermatitis

et al. 2019

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“…The use of vascular laser for fast‐flow lesions has been classically contraindicated as there is a fear of accelerating the progression of the AVM, especially if residual vessels are left untreated. However, there are recent reports of good response to laser treatment in selected patients with Schöbinger stage I AVMs . Both Nd:YAG laser alone and a combination of PDL at 595 nm with Nd‐YAG at 1064 nm (Multiplex system, Cynosure Inc) have been used with up to 90% response rates and no recurrence in acquired digital AVMs .…”

Section: Discussionmentioning

confidence: 99%

Good response to pulsed dye laser in patients with capillary malformation‐arteriovenous malformation syndrome (CM‐AVM)

Iznardo

Roé

Puig

et al. 2020

Pediatric Dermatology

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Capillary malformation‐arteriovenous malformation syndrome (CM‐AVM) is an autosomal dominant disorder caused by heterozygous mutations in RASA1 and EPHB4. Capillary stains in CM‐AVM are compatible with Schöbinger's phase I AVMs. Vascular laser has been classically contraindicated for the treatment of AVMs, as there is a fear of accelerating their progression. In this study, we have treated capillary stains in five CM‐AVM patients with pulsed dye laser, with improvement and without worsening or recurrence of the lesions after 1 year of clinical and ultrasound follow‐up.

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“…Studies of cerebral AVMs have identified the involvement of the vascular endothelial growth factor (VEGF) and plateletderived growth factor (PDGF). However, it is unclear if pathophysiological mechanisms for cerebral and extracranial AVMs are identical [21] . Increased levels of factors involved in vascular destabilization, such as angiopoeitin-2 and matrix metalloproteinase-9, and reduced levels of vascular stabilizers (angiopoeitin-1, integrin, and metalloproteinase inhibitors) have also been found in AVMs [22 , 5] .…”

Section: Etiology and Pathogenesismentioning

confidence: 99%

Management of extracranial arteriovenous malformations of the head and neck

et al. 2020

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The purpose of this study was to review the outcomes of the different therapies for extracranial head and neck arteriovenous malformations (AVMs).AVMs are high-flow congenital vascular anomalies. They are composed of a complex system of vessels directly connecting feeding arteries to draining veins forming a nidus.They may be potentially life-threatening due to progressive symptoms and infiltrative disease. Extracranial AVMs most commonly affect the head and neck area (47.4%) followed by the extremities (28.5%). AVMs are best characterized as being either focal or diffuse. Focal AVMs have good outcomes following adequate treatment. Diffuse lesions have multiple feeding vessel, which results in high rates of recurrence despite treatment.The management of AVMs includes conventional surgery and endovascular techniques. A combination of embolization and surgical resection has become the treatment of choice over the last years. The main goal of both forms of treatment being the complete blockage or resection of the nidus. Transcatheter embolization of vessels has evolved over the years and new embolic agents have emerged. The types of materials available for embolization are classified into mechanical devices, liquid agents and particulates. Efficacy, rate of recurrence and most common complications were evaluated.AVMs recurrence after embolization or resection is reported in up to 80% of cases. Incomplete resection and embolization can induce aggressive growth of the remaining nidus and the risk of progression is up to 50% within the first 5 years and recurrences can occur up to 10 years later.✩ This article was written by members and invitees of the International Head and Neck Scientific Group ( www.IHNSG.com).

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Arteriovenous Malformations: A Diagnostic and Therapeutic Challenge

Cited by 14 publications

References 65 publications

Parkes Weber syndrome presenting as Stewart-Bluefarb acroangiodermatitis

Parkes Weber syndrome presenting as Stewart-Bluefarb acroangiodermatitis

Good response to pulsed dye laser in patients with capillary malformation‐arteriovenous malformation syndrome (CM‐AVM)

Management of extracranial arteriovenous malformations of the head and neck

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