Arteriovenous Anastomoses and Varicose Veins

Gius, John A.

doi:10.1001/archsurg.1960.01300020127019

Cited by 60 publications

(8 citation statements)

References 4 publications

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“…Piulachs and Vidal-Barraquer (1953) put forward the hypothesis that all forms of varicose veins were due to multiple small arterio-venous channels. The work of Gius (1960) and of Haeger and Bergman (1963) supports this hypothesis, but conclusive proof is still needed before it can be accepted without reservation.…”

Section: Resultsmentioning

confidence: 91%

Varicose veins pilot survey, 1966.

Weddell¹

1969

Journal of Epidemiology & Community Health

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Section: Resultsmentioning

confidence: 91%

Varicose veins pilot survey, 1966.

Weddell¹

1969

Journal of Epidemiology & Community Health

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“…Arteriovenous anastomoses have also been demonstrated under direct operative microscopic visualization by Schalin 43 and Gius 44 . In addition, Schroth 45 and Haeger and Bergman 46 have indirectly supported these findings through their evaluations of oxygen content of varicose blood and skin temperature over varicose veins respectively.…”

Section: Pathophysiologymentioning

confidence: 90%

Anatomy and Pathophysiology of Varicose Veins

Goldman

Froněk

1989

The Journal of Dermatologic Surgery and Oncology

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The venous anatomy of the leg is described with special reference to the superficial venous system. The histologic and histochemical differences between varicose and normal veins are presented. The pathophysiology of varicose veins is related to congential or acquired abnormalities of the deep venous system, venous valves, and/or fascial or vein wall weakness. Increased deep venous pressure may be both proximal and distal in etiology, arising from arteriovenous anastomoses, incompetent communicating veins, or venous obstruction. Primary valvular incompetence arises from venous obstruction (thrombosis), thrombophlebitis, or valvular agenesis. Secondary valvular incompetence occurs from deep venous obstruction or increased venous distensibility (usually secondary to circulating estrogens). Finally, fascial weakness of the vein wall or supporting fascia provided a genetic basis for the pathophysiology of varicose veins. Understanding the anatomy and pathophysiology of varicose veins should lead to a more rational and efficacious treatment.

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“…Despite increased capillary blood flow the TcPO 2 of liposclerotic tissue has been demonstrated to be abnormally low 8 . This is in contrast with the PO 2 in the veins of limbs with chronic venous disease that is higher than normal 9–11 . These observations indicate a decreased oxygen release to the tissue, explaining the skin necrosis and ulceration 4 …”

Section: Discussionmentioning

confidence: 99%

Venous Hypertensive Microangiopathy in Relation to Clinical Severity and Effect of Elastic Compression

Christopoulos

Nicolaides

Belcaro

et al. 1991

The Journal of Dermatologic Surgery and Oncology

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Arterial inflow in mL/minute and perimalleolar skin blood flow were measured with venous occlusion air-plethysmography and laser-Doppler flowmetry in 20 normal limbs, 40 limbs with uncomplicated primary varicose veins, 32 limbs with primary varicose veins and skin changes, and 36 limbs with deep venous disease. Limbs with complicated varicose veins (skin changes) and deep venous disease showed a marked increase (2 to 4 times) in both arterial inflow and skin blood flow when compared with the normal controls and uncomplicated varicose veins. The measurements were repeated 30 minutes after the application of graduated elastic stockings. There was a 16% reduction in the mean arterial inflow (P less than .01) and a 45% decrease in skin blood flow (P less than .01). The results indicate that in limbs with chronic venous insufficiency and skin changes there is an increased blood flow which is mainly in the skin. This is irrespective of whether the venous problem is in the superficial or deep venous system. Elastic compression tends to reduce the hyperemia towards the normal range.

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Arteriovenous Anastomoses and Varicose Veins

Cited by 60 publications

References 4 publications

Varicose veins pilot survey, 1966.

Varicose veins pilot survey, 1966.

Anatomy and Pathophysiology of Varicose Veins

Venous Hypertensive Microangiopathy in Relation to Clinical Severity and Effect of Elastic Compression

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