Arterial switch operation: Myocardial ischemia reversed by internal mammary artery graft

Merlo, Maurizio; Brunelli, Federico; Annecchino, Francesco Paolo; Crupi, Giancarlo; Terzi, Amedeo; Ziółkowska, Lidia

doi:10.1016/0003-4975(96)00412-2

Cited by 17 publications

(16 citation statements)

References 9 publications

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“…The theory is supported by the presence of endometriosis in the thoracic cavity and other distant sites outside pelvis as well as detection of endometrial tissue in the uterine vessels in patients with adenomyosis [56]. Lymphovascular metastasis remains a speculative explanation and, while possibly occurring during the development of endometriosis, is not likely to be the primary mechanism as cases of pulmonary and thoracic endometriosis are rare [57,58]. …”

Section: Introductionmentioning

confidence: 99%

Molecular aspects of development and regulation of endometriosis

Aznaurova

Жуматаев

Roberts

et al. 2014

Reprod Biol Endocrinol

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Endometriosis is a common and painful condition affecting women of reproductive age. While the underlying pathophysiology is still largely unknown, much advancement has been made in understanding the progression of the disease. In recent years, a great deal of research has focused on non-invasive diagnostic tools, such as biomarkers, as well as identification of potential therapeutic targets. In this article, we will review the etiology and cellular mechanisms associated with endometriosis as well as the current diagnostic tools and therapies. We will then discuss the more recent genomic and proteomic studies and how these data may guide development of novel diagnostics and therapeutics. The current diagnostic tools are invasive and current therapies primarily treat the symptoms of endometriosis. Optimally, the advancement of “-omic” data will facilitate the development of non-invasive diagnostic biomarkers as well as therapeutics that target the pathophysiology of the disease and halt, or even reverse, progression. However, the amount of data generated by these types of studies is vast and bioinformatics analysis, such as we present here, will be critical to identification of appropriate targets for further study.

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Section: Introductionmentioning

confidence: 99%

Molecular aspects of development and regulation of endometriosis

Aznaurova

Жуматаев

Roberts

et al. 2014

Reprod Biol Endocrinol

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“…In this series, reconstruction of the defect was performed with two distinct patches in all cases. However, excellent results have been demonstrated with the one- or two-patch technique [1, 4, 7, 13]. In our opinion, the two-patch technique is simpler and enables closing the interventricular septal defect with minimal alteration of the valve architecture.…”

Section: Commentmentioning

confidence: 99%

“…Nowadays, primary complete repair is performed once the child has reached the age of 3–6 months [1, 3, 5, 11, 13]. Even in asymptomatic patients, this timing should be respected and the children operated before the age of 1 year because of the development of fixed pulmonary hypertension.…”

Section: Commentmentioning

confidence: 99%

“…Several recent reports describe an improved early and late outcome after surgical repair of the complete atrioventricular canal with a hospital mortality varying between 3 and 10.5% [1, 5, 13, 15]; this represents a considerable step forward, since until the late 1980s, perioperative mortality was found to vary between 5 and 25% [2, 4, 6, 16]. Studer et al [7]found that young age was an incremental risk factor in the early period of their study (1967–1976) but disappeared as risk factor after 1976.…”

Section: Commentmentioning

confidence: 99%

“…Mortality and morbidity are reported to be low, but uncertainty still exists regarding the effect of preoperative conditions on early outcome after surgical repair [1, 2, 3, 4, 5, 6, 7, 8]. In previous series from different institutions, one or several of the following factors were found to be predictors for increased operative mortality and postoperative morbidity: age at operation, severe preoperative regurgitation of the left atrioventricular valve, preoperative heart failure, the presence of accessory atrioventricular valve orifices, additional major intracardiac anomalies, and the presence of Down’s syndrome [3, 7, 8, 9].…”

Section: Introductionmentioning

confidence: 99%

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Surgery of the Complete Atrioventricular Canal: Relationship between Age at Operation, Mitral Regurgitation, Size of the Ventricular Septum Defect, Additional Malformations and Early Postoperative Outcome

Schaffer

Berdat²,

Stolle³

et al. 1999

Cardiology

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The perioperative risk after repair of the complete atrioventricular canal is reported to be low provided there is a balanced relationship of both ventricles, and elevated pulmonary vascular resistance has been demonstrated to be reversible. However, some pre- and perioperative conditions may adversely affect early postoperative outcome. A consecutive series of 42 patients (mean age 8.5 months, 34/42, 81%, with trisomy 21) operated between 1994 and 1998 was analyzed prospectively for pre- and perioperative risk factors. Echocardiography alone was performed in 35 patients whereas cardiac catheterization was performed in 7 patients aged more than 18 months to confirm operability. Additional malformations were found in 20 patients. Early mortality was 2.3% (1/42), and postoperatively a low cardiac output was observed in 25 patients (59%); NO inhalation was used in 12 patients. Only the size of the ventricular septal defect (> than the diameter of the aortic annulus) could be identified as a predictor of adverse postoperative outcome in multivariate analysis. The presence of associated intracardiac malformations showed a trend to increased perioperative risk in multivariate analysis. Early results after repair of the complete atrioventricular canal are excellent. Survival, postoperative morbidity and normalization of pulmonary artery pressure can be related to the size of the ventricular septal defect. Continuous monitoring of the pulmonary artery pressure (with consequent NO application when indicated) is probably responsible for the low perioperative risk encountered in this series, despite the high incidence of postoperative pulmonary artery hypertension.

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Coronary Artery Involvement of Williams Syndrome in Infants and Surgical Revascularization Strategy

Federici

Ranghetti

Merlo

et al. 2016

The Annals of Thoracic Surgery

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Williams syndrome (WS) is a genetic disorder due to deficiency of elastin gene expression. It is characterized by typical somatic abnormalities and a wide range of cardiovascular malformations. Coronary artery involvement is a frequent finding of the syndrome, particularly in those patients with severe supravalvular aortic stenosis. We present the case of an 11-month-old infant affected by WS who developed severe coronary artery disease 2 months after the surgical repair of supravalvular aortic stenosis. The clinical picture and successful surgical revascularization strategy is also described.

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Arterial switch operation: Myocardial ischemia reversed by internal mammary artery graft

Cited by 17 publications

References 9 publications

Molecular aspects of development and regulation of endometriosis

Molecular aspects of development and regulation of endometriosis

Surgery of the Complete Atrioventricular Canal: Relationship between Age at Operation, Mitral Regurgitation, Size of the Ventricular Septum Defect, Additional Malformations and Early Postoperative Outcome

Coronary Artery Involvement of Williams Syndrome in Infants and Surgical Revascularization Strategy

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