Arterial bending angle and wall morphology correlate with slow coronary flow: Determination with multidetector CT coronary angiography

Kantarcı, Mecit; Gündoğdu, Fuat; Doğanay, Selim; Duran, Cihan; Kalkan, Mehmet; Sagsoz, M. Erdem; Küçük, Osman; Karakaya, Afak Durur; Küçük, Ahmet; Akgün, Metin

doi:10.1016/j.ejrad.2009.07.002

Cited by 14 publications

(12 citation statements)

References 17 publications

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“…This study also found the almost similar incidence of multiple myocardial bridges compared with the studies done by (Kosiński and Grzybiak, 2001;Loukas et al, 2006;Nalinakumari et al, 2015). Coronary artery tortuosity caused by myocardial bridges associated with chest pain, slow low coronaries and compromised blood low and atherosclerosis reported by Gaibazzi et al (2011); Kantarci et al (2011); An et al (2018).…”

Section: Resultsmentioning

confidence: 98%

Hair pin bends of epicardial coronary arteries - a unique feature in multiple myocardial bridges

G.V¹,

Panda²

2019

ijrps

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Myocardial bridge(MB) is an anatomical variant. Sometimes MB can cause compression over the coronary arteries and causes ischemic heart diseases.MB associated with bends of coronary arteries double the risk of coronary artery disease. So the present study aimed to find out the incidence of bends of the coronary artery along with the myocardial bridging. 100 heart specimens were obtained from routine dissection conducted for undergraduate students in the department of Anatomy IMS & SUM Hospital Bhubaneswar. After the simple dissecting procedure, epicardial coronary arteries, their branches and myocardial bridges and hairpin bends of coronary arteries were observed. Myocardial bridges present 41 (41%) over Left anterior descending artery(LAD) only.Among 41 hearts single myocardial bridge present in 37((90.25%) hearts, double myocardial bridge were present in 3((7.31%) hearts and triple myocardial bridges present in only one heart(2.44%). Hairpin bends of the coronary artery were present in double and triple myocardial bridged hearts only. No hairpin bends of the coronary artery was observed in the single myocardial bridge. Hair pin bends of the coronary arteries are the unique features of the myocardial bridges in multiple myocardial bridges, i.e. double and triple myocardial bridges. However, hairpin bends of coronary arteries were absent in single myocardial bridged hearts.

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Section: Resultsmentioning

confidence: 98%

Hair pin bends of epicardial coronary arteries - a unique feature in multiple myocardial bridges

G.V¹,

Panda²

2019

ijrps

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“…Recent studies have found that anatomical characteristics of coronary arteries and thickness of subpericardial fat correlated with occurrence of CMVD [17, 18]. Multiple clinical studies have shown that most risk factors for coronary atherosclerosis were also risk factors for CMVD, such as smoking, being male, hyperhomocysteinemia, abnormal glucose tolerance, diabetes, hypersensitive CRP, hyperlipidemia, and other independent predictors of CMVD [19].…”

Section: Discussionmentioning

confidence: 99%

Do Statins Have a Positive Impact on Patients with Coronary Microvascular Dysfunction on Long-Term Clinical Outcome? A Large Retrospective Cohort Study

Luo

Guo

Huang

et al. 2019

BioMed Research International

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Objectives. To investigate the influence of statins on major adverse cardiovascular events (MACE) in patients with coronary microvascular dysfunction (CMVD). Participants. 23,494 patients who received coronary angiography (CAG) were included. Thrombolysis in Myocardial Infarction, Myocardial Perfusion Grading (TMPG), a useful angiographic method, was used to evaluate CMVD. Results. Using multivariate analysis, NYHA III/IV (HR, 1.44; 95% CI, 1.03-2.01; P=0.031), PCI history (HR, 3.69; 95% CI, 2.57-5.31; P<0.001), TG (HR, 1.15; 95% CI, 1.06-1.26; P=0.001), creatinine (HR, 1.00; 95% CI, 1.00-1.01; P<0.001), cTnT (HR, 0.98; 95% CI, 0.96-0.99; P<0.001), heart rate (HR, 0.98; 95% CI, 0.97-0.99; P=0.001), β-blocker (HR, 0.68; 95% CI, 0.51-0.91; P=0.008), aspirin (HR, 0.38; 95% CI, 0.24-0.61; P<0.001), and statins (HR, 0.33; 95% CI, 0.19-0.60; P<0.001) significantly correlated with reduced MACE in CMVD patients. In subgroups analysis, statins decreased MACE overall (HR, 0.33; 95% CI, 0.19-0.59; P<0.001) and in CMVD patients with smoking history (HR, 0.64; 95% CI, 0.43-0.93; P=0.014), diabetes (HR,0.27; 95% CI,0.12-0.61; P=0.002), hypertension (HR, 0.10; 95% CI, 0.03-0.36; P=0.001), and hypertension and diabetes (HR, 0.09; 95% CI, 0.014-0.53; P=0.008). Conclusion. Statins could reduce MACE in patients with CMVD.

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“…16,17 Using CT coronary angiography it can be seen that in patients with CSFP, the angulation of the main coronary arteries of the aorta is seen to be smaller. 18 Katarci et al conducted a case-control study to examine the correlation between the anatomic properties of the coronary arteries and the incidence of CSFP. The results showed that CSFP was associated with higher tortuosity and more distal branches of the coronary arteries.…”

Section: Anatomical Factorsmentioning

confidence: 99%

Role of Fibroblast Growth Factor-23 in Coronary Slow Flow Phenomenon Pathogenesis

Oktaviandani

Indrajaya

Ghanie

et al. 2021

bsm

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The phenomenon of angina chest pain without significant epicardial coronary artery stenosis, but accompanied by a slowdown in coronary blood flow is often found in patients with symptoms of acute coronary syndrome who undergoing invasive coronary angiography. This phenomenon of slow coronary blood flow is then called the coronary slow flow phenomenon (CSFP). The pathogenesis mechanism of CSFP remains unclear. The pathogenesis of CSFP is thought to be multifactorial. Endothelial dysfunction, small vessel disease, inflammation, renin system angiotensin aldosterone, atherosclerosis are thought to be involved in the pathogenesis of CSFP. Cardiovascular disease incidence and death were associated with elevated levels of Fibroblast growth factor-23 (FGF-23). High levels of FGF-23 can lead to formation of blood vessel calcification, left ventricular hypertrophy, arterial stiffness, endothelial dysfunction, increased inflammatory markers and elevated levels of angiotensin II. It is suspected that FGF-23 has a role in this event other than as a regulator of bone and mineral metabolism. This literature review aims to determine the relationship between fibroblast growth factor-23 and the pathophysiology of CSFP. Based on the broad role of FGF-23, it is possible that FGF-23 is involved in the pathogenesis of CSFP.

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Arterial bending angle and wall morphology correlate with slow coronary flow: Determination with multidetector CT coronary angiography

Cited by 14 publications

References 17 publications

Hair pin bends of epicardial coronary arteries - a unique feature in multiple myocardial bridges

Hair pin bends of epicardial coronary arteries - a unique feature in multiple myocardial bridges

Do Statins Have a Positive Impact on Patients with Coronary Microvascular Dysfunction on Long-Term Clinical Outcome? A Large Retrospective Cohort Study

Role of Fibroblast Growth Factor-23 in Coronary Slow Flow Phenomenon Pathogenesis

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