Are rises in Electro-Magnetic Field in the human environment, interacting with multiple environmental pollutions, the tipping point for increases in neurological deaths in the Western World?

Pritchard, Colin; Silk, Anne; Hansen, Lars

doi:10.1016/j.mehy.2019.03.018

Cited by 21 publications

(22 citation statements)

References 106 publications

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“…As a consequence of that, people circa age 30 have been coming down with AD – these cases are still relatively rare, but they were unheard of until recently. One study [ 26 ] suggests EMF causation of such early onset.…”

Section: Decreasing Age Of Onset Of Admentioning

confidence: 99%

Low Intensity Electromagnetic Fields Act via Voltage-Gated Calcium Channel (VGCC) Activation to Cause Very Early Onset Alzheimer’s Disease: 18 Distinct Types of Evidence

Pall

2022

CAR

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Electronically generated electromagnetic fields (EMFs) including those used in wireless communication such as cell phones, Wi-Fi and smart meters, are coherent, producing very high electric and magnetic forces which act on the voltage sensor of voltage-gated calcium channels to produce increases in intracellular calcium [Ca2+]i. The calcium hypothesis of Alzheimer’s disease (AD) has shown that each of the important AD-specific and nonspecific causal elements are produced by excessive [Ca2+]i. [Ca2+]i acts in AD via excessive calcium signaling and the peroxynitrite/oxidative stress/inflammation pathway which are each elevated by EMFs. An apparent vicious cycle in AD involves amyloid-beta protein (A) and [Ca2+]i. Three types of epidemiology each suggest EMF causation of AD including early onset AD. Extensive animal model studies show that low intensity EMFs cause neurodegeneration including AD, with AD animals having elevated levels of A, amyloid precursor protein and BACE1. Rats exposed to pulsed EMFs every day are reported to develop universal or near universal very very very early onset neurodegeneration including AD; these findings are superficially similar to humans with digital dementia. EMFs producing modest increases in [Ca2+]i can also produce protective, therapeutic effects. The therapeutic pathway and peroxynitrite pathway inhibit each other. A summary of 18 different findings is provided, which collectively provide powerful evidence for EMF causation of AD. The author is concerned that smarter, more highly pulsed “smart” wireless communication may cause widespread very, very early onset AD in human populations.

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Section: Decreasing Age Of Onset Of Admentioning

confidence: 99%

Low Intensity Electromagnetic Fields Act via Voltage-Gated Calcium Channel (VGCC) Activation to Cause Very Early Onset Alzheimer’s Disease: 18 Distinct Types of Evidence

Pall

2022

CAR

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“…Frequencies in the range of the visible light are central for the homeostasis of the circadian rhythms of the nervous system and metabolism of the whole body [1,2]. On the other side unnatural microwave frequencies of modern wireless technologies (smartphones, cell towers and WiFi/WLAN) have devastating biological effects which are clearly proven by hundreds of scientific publications worldwide [3][4][5][6][7][8][9][10]. However, we can use frequencies of the ultraviolet (UV) spectrum for therapeutic purposes.…”

Section: Introductionmentioning

confidence: 99%

Replacement of 8-MOP/UV-A treatment by UV-C irradiation for further development of Extracorporeal Photopheresis

Budde¹,

Zwerenz²,

Barrios-Bussmann³

et al. 2021

Hematol Med Oncol

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Extracorporeal photopheresis (ECP) has been used for more than 30 years to treat inflammatory disorders such as graft-versus-host disease (GvHD). The central step is the ex vivo treatment of the patient´s leukocytes with 8-Methoxypsoralen (8-MOP) and UV-A irradiation, resulting in cellular apoptosis. However, the injection of 8-MOP into the irradiation bag poses a risk of contamination. UV-C radiation is more energetic than UV-A radiation and is able to induce apoptosis without photosensitizers such as 8-MOP. Hence, we compared the effects of both treatments on leukocytes in vitro and in vivo. We analyzed the cell-specific pattern of apoptosis in human mononuclear cells treated with 8-MOP/UV-A and UV-C in cell culture for 72 hours. In addition, we investigated the therapeutic potential of UV-C irradiated leukocytes in a mouse model of acute GvHD. In vitro we observed equal or only slightly decreased levels of apoptosis in cells treated with UV-C compared to 8-MOP/UV-A. Both, the kinetic and final level of apoptosis after 72 h were similar in T cells, monocytes, B cells, regulatory T cells and NK(T) cells. The therapeutic in vivo analysis in a murine GvHD model revealed no clear benefit of the UV-C treatment compared to the non-therapeutic control group, but also no adverse survival or clinical GVHD score compared to the 8-MOP/UV-A group. Our in vitro and in vivo data support the notion that 8-MOP/UV-A could be replaced by UV-C treatment in ECP and thus contribute to a safer GvHD therapy. The investigation of the therapeutic in vivo potential should be continued in further studies.

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“…Household appliances and other electrical devices, as well as power transmission networks and mobile antennas create electromagnetic fields, significantly increase the electromagnetic radiation in the human environment [1,2]. There have been many discussions about microwave ovens, computers, transmitters of various communication devices, and other EMF sources that influence people, especially children's, health.…”

Section: Introductionmentioning

confidence: 99%

Investigation of the Possibilities of Wool Fiber Surface Modification with Copper Selenide

2021

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A study of altering the conductive properties of wool fibers by applying copper selenide is presented. The researched modification of wool fibers was based on a two-stage adsorption-diffusion process. X-ray diffraction, scanning electron microscope, energy-dispersive X-ray spectrum, and Fourier transform infrared spectroscopy were performed to evaluate the morphological and physical characteristics of all CuxSe-coated wool fibers. X-ray diffraction (XRD) data showed a single, Cu0.87Se (klockmannite), crystalline phase present, while Atomic Absorption Spectroscopy (AAS) and Energy Dispersive X-ray (EDX) analyses showed that the concentrations of Cu and Se in copper selenide coatings depend on the number of wool fiber treatment cycles. It was determined that a dense layer of CuxSe grows through a nucleation mechanism followed by particle growth to fill out the complete surface. It was found that the conductivity of the coated wool fibers depends on the quality and density of the copper selenide coating, thus the resistance of electrically impermeable wool fibers can be reduced to 100 Ω by increasing the number of treatment cycles.

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Are rises in Electro-Magnetic Field in the human environment, interacting with multiple environmental pollutions, the tipping point for increases in neurological deaths in the Western World?

Cited by 21 publications

References 106 publications

Low Intensity Electromagnetic Fields Act via Voltage-Gated Calcium Channel (VGCC) Activation to Cause Very Early Onset Alzheimer’s Disease: 18 Distinct Types of Evidence

Low Intensity Electromagnetic Fields Act via Voltage-Gated Calcium Channel (VGCC) Activation to Cause Very Early Onset Alzheimer’s Disease: 18 Distinct Types of Evidence

Replacement of 8-MOP/UV-A treatment by UV-C irradiation for further development of Extracorporeal Photopheresis

Investigation of the Possibilities of Wool Fiber Surface Modification with Copper Selenide

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