Approaches for identifying and defining environmentally associated rheumatic disorders

Miller, Frederick W.; Hess, Evelyn V.; Clauw, Daniel J.; Hertzman, Phillip A.; Pincus, Theodore; Silver, Richard M.; Mayes, Maureen D.; Varga, John; Medsger, Thomas A.; Love, Lori A.

doi:10.1002/1529-0131(200002)43:2<243::aid-anr2>3.0.co;2-k

Cited by 85 publications

(35 citation statements)

References 37 publications

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“…According to proposed criteria 11 , an illness, like neuropathy, can be considered as drug-induced or environmentally associated based on: [1] temporal closed association; [2] lack of likely alternative explanations; [3] stabilization or improvement by interrupting exposure to the presumed inciting agent; [4] rechallenge; [5] biological plausibility; [6] analogy; [7] dose responsiveness; [8] specificity. To report findings of a possible causal relationship between exposure and clinical syndrome, at least 4 of the 8 attribution elements should be present.…”

Section: Motor Neuropathymentioning

confidence: 99%

Motor neuropathy with multiple conduction blocks associated with TNF-alpha antagonist

Carrilho

Araújo

Alves

et al. 2010

Arq. Neuro-Psiquiatr.

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Section: Motor Neuropathymentioning

confidence: 99%

Motor neuropathy with multiple conduction blocks associated with TNF-alpha antagonist

Carrilho

Araújo

Alves

et al. 2010

Arq. Neuro-Psiquiatr.

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“…In addition to developing standard assessment criteria, the study group also recommended a four stage process of identifying and defining environmentally associated rheumatologic disorders (Miller et al, 2000). During stage 1, an association is proposed via case reports or case series.…”

Section: Acknowledgementsmentioning

confidence: 99%

“…The study group recognized that, in reality, research activities may occur in several stages simultaneously. However, the stage system provides explicit consideration of the amount of evidence supporting an association, thus preventing the premature (or delayed) acceptance of a causal association relative to the weight of the evidence (Miller, 2000). Since their publication, these study group criteria have been utilized to evaluate the relationship between anti-tumor necrosis factor α therapy for inflammatory arthritides and demyelination events (Mohan, 2001), and have been cited by an number of authors evaluating the relationship between environmental factors and rheumatologic illness (e.g., Nietert & Silver, 2000; Bresnihan & Cunnane 2003).…”

Section: Acknowledgementsmentioning

confidence: 99%

“…These standardized methods were created because of the "real world" need to have standardized methods for determining causation (e.g., Is there sufficient evidence from case reports and other data to determine that a drug causes a particular adverse event?). Recently, the American College of Rheumatology convened an Environmental Disease Study Group to develop consensus standards for the process of identifying rheumatologic diseases that result from environmental exposures (Miller et al, 2000). In developing their assessment criteria, the study group examined contemporary algorithms for determining causation, and used the relative prevalence of individual criteria currently in use to develop their own consensus criteria (Table II).…”

mentioning

confidence: 99%

“…Recently, the American College of Rheumatology convened an Environmental Disease Study Group to develop consensus standards for the process of identifying rheumatologic diseases that result from environmental exposures (Miller et al, 2000). In developing their assessment criteria, the study group examined contemporary algorithms for determining causation, and used the relative prevalence of individual criteria currently in use to develop their own consensus criteria (Table II).In addition to developing standard assessment criteria, the study group also recommended a four stage process of identifying and defining environmentally associated rheumatologic disorders (Miller et al, 2000). During stage 1, an association is proposed via case reports or case series.…”

mentioning

confidence: 99%

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Mechanisms in Chronic Multisymptom Illnesses

Clauw¹

2008

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Public reporting burden for this collection of information is estimated to average 1 hour per response, including the time for reviewing instructions, searching existing data sources, gathering and maintaining the data needed, and completing and reviewing this collection of information. Send comments regarding this burden estimate or any other aspect of this collection of information, including suggestions for reducing this burden to Department of Defense, Washington Headquarters Services, Directorate for Information Operations and Reports (0704-0188), 1215 Jefferson Davis Highway, Suite 1204, Arlington, VA 22202-4302. Respondents should be aware that notwithstanding any other provision of law, no person shall be subject to any penalty for failing to comply with a collection of information if it does not display a currently valid OMB control number. PLEASE DO NOT RETURN YOUR FORM TO THE ABOVE ADDRESS. 5e. TASK NUMBER 5f. WORK UNIT NUMBER REPORT DATE 01-06-2006 REPORT TYPE PERFORMING ORGANIZATION NAME(S) AND ADDRESS(ES) 8. PERFORMING ORGANIZATION REPORT NUMBERUniversity of Michigan Ann Arbor, MI 48106-0737 SPONSORING / MONITORING AGENCY NAME(S) AND ADDRESS(ES) 10. SPONSOR/MONITOR'S ACRONYM(S) U.S. Army Medical Research and Materiel Command Fort Detrick, Maryland 21702-5012 SPONSOR/MONITOR'S REPORT NUMBER(S) DISTRIBUTION / AVAILABILITY STATEMENTApproved for Public Release; Distribution Unlimited SUPPLEMENTARY NOTESOriginal contains colored plates: ALL DTIC reproductions will be in black and white. ABSTRACTThe overall objectives of this cooperative agreement are to conduct research in pursuit of identifying the physiologic mechanisms responsible for the symptoms of pain, fatigue, and memory difficulties commonly seen in patients with Chronic Multisymptom Illnesses (CMI) (i.e., fibromyalgia, chronic fatigue syndrome, Gulf War Illnesses, etc.); to identify the risk factors for developing these syndromes as well as programs aimed at both preventing these illnesses and treating established cases. These objectives will be achieved through multiple research studies using innovative, technologically advanced (e.g., functional MRI and telemedicine) methodologies in a multidisciplinary environment. Various studies will be conducted to explore all aspects of pain processing, the effects of exercise deprivation and sleep reduction on symptomatology, the ability of exercise and/or cognitive behavioral therapies to alter patients' locus of control for pain, the neurobiological mechanism(s) of acupuncture on analgesia, the presence of hypersensitivity to auditory stimuli, and the effectiveness of cognitive behavioral therapy delivered via telemedicine and the internet. These studies will be conducted on well-characterized cohorts of CMI subjects and healthy controls taken from our burgeoning subject registry. Research will occur over the next 3 years at the University of Michigan, Ann Arbor, MI and Avera Research Institute, Sioux Falls, SD. SUBJECT TERMS INTRODUCTION:Researchers in the Chronic Pain and Fatigue Research Center ...

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Rigorous New Approach to Constructing a Gold Standard for Validating New Diagnostic Criteria, as Exemplified by the Eosinophilia-Myalgia Syndrome

Hertzman¹,

Clauw²,

Duffy³

et al. 2001

Arch Intern Med

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Approaches for identifying and defining environmentally associated rheumatic disorders

Cited by 85 publications

References 37 publications

Motor neuropathy with multiple conduction blocks associated with TNF-alpha antagonist

Motor neuropathy with multiple conduction blocks associated with TNF-alpha antagonist

Mechanisms in Chronic Multisymptom Illnesses

Rigorous New Approach to Constructing a Gold Standard for Validating New Diagnostic Criteria, as Exemplified by the Eosinophilia-Myalgia Syndrome

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