Application of the evoked response technique in air pollution toxicology

Xintaras, Charles

doi:10.1016/0041-008x(66)90103-7

Cited by 24 publications

(2 citation statements)

References 5 publications

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“…The present study demonstrated that exposure of conscious rats to 0.5 and 1.0 ppm O3 produces marked decreases in HR, decreases in W with concomitant increases in SWS, sustained suppression of PS and decreases in powers of high frequency bands (8)(9)(10)(11)(12)(13)(14)(15)(16)(17)(18)(19)(20)(21)(22)(23)(24) Figure 5. Effects of atropine on HR (top), amounts of W, SWS and PS (middle) and power values of the parietal EEG spectra (bottom).…”

Section: Discussionmentioning

confidence: 99%

Acute Effects of Ozone on EEG Activity, Sleep-Wakefulness and Heart Rate in Rats.

1992

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Abstract:Effects of exposure to O3 on EEG activity, sleep-wakefulness and heart rate were examined using conscious rats which had been chronically implanted with electrodes for EEG, EMG and ECG recordings. Exposure to 0.5 ppm O3 for 6 hrs and 1.0 ppm O3 for 3 hrs suppressed amounts of wakefulness (W) and paradoxical sleep (PS) at the expense of an increase in slow-wave sleep (SWS), and lowered the amplitude of fast EEG waves and heart rate (HR). The lowered EEG amplitude and the suppressed PS recovered more rapidly during the post-exposure period than did the lowered HR. The ip administration of atropine sulfate blocked the suppressed W, the increased SWS and the lowered HR, while the lowered EEG amplitude and the suppressed PS were not blocked. These observations suggest that the O3-induced bradycardia results from enhanced activity of cardiac parasympathetic nerves and that the O3-induced changes in W and SWS result secondarily from some circulatory factor including the bradycardia.

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Section: Discussionmentioning

confidence: 99%

Acute Effects of Ozone on EEG Activity, Sleep-Wakefulness and Heart Rate in Rats.

1992

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“…Previous work from our laboratory indicated that the evoked response may be a sensitive and reliable measure of the effect of exposure to carbon monoxide and ozone. 1 Despite frequent disagreement regarding the origin and meaning of evoked potentials, it is generally agreed that different neural elements sequentially activated will be reflected in the sequential appearance of various components of the waveform of the evoked potential. Stated differently, it is assumed that different phases of the evoked response arise as a result of the different pathways along which the afferent impulse enters the cerebral cortex or other subcortical receiving area.…”

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confidence: 99%

Brain Potentials Studied by Computer Analysis

Xintaras¹,

Ce²,

Mf³

et al. 1966

Archives of Environmental Health: An International Journal

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Kohlenoxid [MAK Value Documentation in German language, 1975]

2012

The MAK‐Collection for Occupational Health and Safety

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Veröffentlicht in der Reihe Gesundheitsschädliche Arbeitsstoffe , 4. Lieferung, Ausgabe 1975 Der Artikel enthält folgende Kapitel: Allgemeiner Wirkungscharakter Erfahrungen beim Menschen Tierexperimentelle Befunde Begründung des MAK‐Wertes

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Application of the evoked response technique in air pollution toxicology

Cited by 24 publications

References 5 publications

Acute Effects of Ozone on EEG Activity, Sleep-Wakefulness and Heart Rate in Rats.

Acute Effects of Ozone on EEG Activity, Sleep-Wakefulness and Heart Rate in Rats.

Brain Potentials Studied by Computer Analysis

Kohlenoxid [MAK Value Documentation in German language, 1975]

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