Aphasia in Subdural Hematoma

Dell, Stephen O.; Batson, Ramon; Kasdon, David L.; Peterson, Thomas

doi:10.1001/archneur.1983.04050030071015

Cited by 12 publications

(7 citation statements)

References 31 publications

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“…Rarely, aphasia can also be a presenting symptom of subdural haematomas [5,6]. However, to our knowledge, Gerstmann' s syndrome has not been previously reported.…”

Section: Introductionmentioning

confidence: 79%

Case Study: Gerstmann's syndrome associated with chronic subdural haematoma: a case report

Okumura

1998

Brain Injury

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We report a patient who exhibited Gerstmann's syndrome in association with a chronic subdural haematoma. A 71-year-old right-handed woman presented with mild right arm and leg weakness that began 2 weeks prior to admission. Neurological examination on admission revealed a mild right hemiparesis. Neuropsychological examination revealed right-left disorientation, finger agnosia, agraphia, and acalculia, but no language disturbance. A computerized tomographic (CT) scan revealed a large left frontoparietal, extra-axial hypodense fluid collection containing scattered hypodense foci. A left parietal evacuation of the haematoma was performed. Following surgery the patient dramatically improved. We suggest that the direct compression by the chronic subdural haematoma or a hemispheric pressure difference caused Gerstmann's syndrome. This is an unusual report of a Gerstmann's syndrome following chronic subdural haematoma.

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“…Rarely, aphasia can also be a presenting symptom of subdural haematomas [5,6]. However, to our knowledge, Gerstmann' s syndrome has not been previously reported.…”

Section: Introductionmentioning

confidence: 79%

Case Study: Gerstmann's syndrome associated with chronic subdural haematoma: a case report

Okumura

1998

Brain Injury

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“…They can be challenging clinically. The absence of significant other neurological signs can result in cases being misdiagnosed as a transient ischemic attack or cerebrovascular accident [3]. They can be dismissed as intoxication, confusion, and psychosis or even malingering.…”

Section: Discussionmentioning

confidence: 99%

“…It is most commonly associated with right blunt orbito-frontal trauma with a contre-coup left temporo-parietal injury [3, 11, 12]. This type of impact imparts a shearing force to the head which causes motion of the cerebral hemispheres within the closed cranium.…”

Section: Discussionmentioning

confidence: 99%

Variations in the Presentation of Aphasia in Patients with Closed Head Injuries

Kavanagh

Lynam

Düerk

et al. 2010

Case Reports in Medicine

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Impairments of speech and language are important consequences of head injury as they compromise interaction between the patient and others. A large spectrum of communication deficits can occur. There are few reports in the literature of aphasia following closed head injury despite the common presentation of closed head injury. Herein we report two cases of closed head injuries with differing forms of aphasia. We discuss their management and rehabilitation and present a detailed literature review on the topic. In a busy acute surgical unit one can dismiss aphasia following head injury as behaviour related to intoxication. Early recognition with prolonged and intensive speech and language rehabilitation therapy yields a favourable outcome as highlighted in our experience. These may serve as a reference for clinicians faced with this unusual outcome.

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“…An unusual patient reported by Fowler [3] had frequent TIA-like symptoms during 44 years prior to the diagnosis of meningioma. Recurrent neurological deficit in the form of transient global amnesia has recently been reported in association with temporal meningioma [4], TIA-like epi sodes may also be caused by chronic subdural hemato mas [5,6], Meningioma may even cause recurrent tran sient focal neurological signs, simulating retrobulbar neuritis [7], Several mechanisms have been suggested for the pathophysiology of these 'transient tumor attacks', such as local pressure of the tumor, Todd's paralysis and vas cular insufficiency. This could be due to vasospasm sim ilar to migraine [1], 'steal syndrome' or alteration of blood flow due to dynamic changes in the extent of local brain edema [8], A popular explanation is the spreading cortical depression theory introduced by Ledo [9], One may also consider the simultaneous occurrence of classical TIA and meningioma in the same individual.…”

Section: Discussionmentioning

confidence: 99%