Anxiety Sensitivity Prospectively Predicts Posttraumatic Stress Symptoms Following a Campus Shooting

Boffa, Joseph W.; Norr, Aaron M.; Raines, Amanda M.; Albanese, Brian J.; Short, Nicole A.; Schmidt, Norman B.

doi:10.1016/j.beth.2016.02.006

Cited by 55 publications

(50 citation statements)

References 44 publications

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“…Results further indicated that ASCC was more strongly related to PTS among those with a greater number of probable TBIs, indicating that a TBIs may potentiate the effects of variability in ASCC on PTS symptomology. These results are consistent with prior findings demonstrating that ASCC (Asmundson & Stapleton, 2008; Boffa et al, 2016; Vujanovic et al, 2008) and probable TBIs (Bryan & Clemans, 2013; Ruff et al, 2012; Schneiderman et al, 2008) contribute to PTS symptoms, and extend our knowledge of these relationships by demonstrating a two-way interaction of ASCC and the number of probable TBIs an individual has incurred.…”

Section: Discussionsupporting

confidence: 91%

“…Olatunji and Fan (2015) utilized a trauma-film lab paradigm to elucidate the role of ASCC in PTS symptoms and found that ASCC predicted greater trauma-related intrusive thoughts throughout 1 week following the trauma-film, providing further evidence for the causal role of ASCC in PTS symptomology. This finding was supported in a naturalistic longitudinal study in which ASCC prior to a traumatic event (i.e., a campus shooting) prospectively predicted post-shooting PTS symptoms (Boffa et al, 2016). Taken together, extant literature strongly supports the role of ASCC in the etiology and maintenance of PTS symptoms.…”

Section: Introductionmentioning

confidence: 71%

“…As described above, ASCC increases PTS through the amplification of cognitive-dyscontrol symptoms of anxiety through the misinterpretation of these sensations as harmful (Asmundson & Stapleton, 2008; Boffa et al, 2016; Taylor, 2003; Vujanovic et al, 2008). Importantly, TBI is associated with similar symptoms of cognitive dyscontrol (Ryan & Warden, 2003) which may be amplified by AS (Albanese, Boffa, Macatee, & Schmidt, 2017) and are theorized to be a mechanism explaining the link between TBI and PTS (Aupperle, Melrose, Stein, & Paulus, 2012).…”

Section: Introductionmentioning

confidence: 91%

“…Several studies have demonstrated a positive relationship between ASCC and PTS (Asmundson & Stapleton, 2008; Boffa et al, 2016; Vujanovic, Zvolensky, & Bernstein, 2008). For example, active-duty police officers with PTSD have greater ASCC compared to active-duty police officers without PTSD (Asmundson & Stapleton, 2008).…”

Section: Introductionmentioning

confidence: 99%

“…Given extant research associating PTS symptoms with higher ASCC (Asmundson & Stapleton, 2008; Boffa et al, 2016; Vujanovic et al, 2008) and a greater number of TBIs (Barnes et al, 2012; Bryan & Clemans, 2013; Bryant et al, 2010; Hoge et al, 2008; Ruff et al, 2012; Schneiderman et al, 2008), we hypothesized that both of these factors would demonstrate unique and positive relationships with PTS symptoms. Moreover, given evidence that ASCC amplifies PTS through the exacerbation of anxiety-related cognitive dyscontrol symptoms, we hypothesized that ASCC would moderate the effects of increasing numbers of probable TBIs on elevated PTS such that the effects of TBI would be stronger among those with greater ASCC.…”

Section: Introductionmentioning

confidence: 99%

See 4 more Smart Citations

Interactive Effects of Traumatic Brain Injury and Anxiety Sensitivity Cognitive Concerns on Post-traumatic Stress Among Active Duty Soldiers

Albanese¹,

Macatee²,

Schmidt³

et al. 2017

Cogn Ther Res

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Section: Discussionsupporting

confidence: 91%

Section: Introductionmentioning

confidence: 71%

Section: Introductionmentioning

confidence: 91%

Section: Introductionmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 99%

See 3 more Smart Citations

Interactive Effects of Traumatic Brain Injury and Anxiety Sensitivity Cognitive Concerns on Post-traumatic Stress Among Active Duty Soldiers

Albanese¹,

Macatee²,

Schmidt³

et al. 2017

Cogn Ther Res

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Utility of Wrist-Wearable Data for Assessing Pain, Sleep, and Anxiety Outcomes After Traumatic Stress Exposure

Straus

et al. 2023

JAMA Psychiatry

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ImportanceAdverse posttraumatic neuropsychiatric sequelae after traumatic stress exposure are common and have higher incidence among socioeconomically disadvantaged populations. Pain, depression, avoidance of trauma reminders, reexperiencing trauma, anxiety, hyperarousal, sleep disruption, and nightmares have been reported. Wrist-wearable devices with accelerometers capable of assessing 24-hour rest-activity characteristics are prevalent and may have utility in measuring these outcomes.ObjectiveTo evaluate whether wrist-wearable devices can provide useful biomarkers for recovery after traumatic stress exposure.Design, Setting, and ParticipantsData were analyzed from a diverse cohort of individuals seen in the emergency department after experiencing a traumatic stress exposure, as part of the Advancing Understanding of Recovery After Trauma (AURORA) study. Participants recruited from 27 emergency departments wore wrist-wearable devices for 8 weeks, beginning in the emergency department, and completed serial assessments of neuropsychiatric symptoms. A total of 19 019 patients were screened. Of these, 3040 patients met study criteria, provided informed consent, and completed baseline assessments. A total of 2021 provided data from wrist-wearable devices, completed the 8-week assessment, and were included in this analysis. The data were randomly divided into 2 equal parts (n = 1010) for biomarker identification and validation. Data were collected from September 2017 to January 2020, and data were analyzed from May 2020 to November 2022.ExposuresParticipants were recruited for the study after experiencing a traumatic stress exposure (most commonly motor vehicle collision).Main Outcomes and MeasuresRest-activity characteristics were derived and validated from wrist-wearable devices associated with specific self-reported symptom domains at a point in time and changes in symptom severity over time.ResultsOf 2021 included patients, 1257 (62.2%) were female, and the mean (SD) age was 35.8 (13.0) years. Eight wrist-wearable device biomarkers for symptoms of adverse posttraumatic neuropsychiatric sequelae exceeded significance thresholds in the derivation cohort. One of these, reduced 24-hour activity variance, was associated with greater pain severity (r = −0.14; 95% CI, −0.20 to −0.07). Changes in 6 rest-activity measures were associated with changes in pain over time, and changes in the number of transitions between sleep and wake over time were associated with changes in pain, sleep, and anxiety. Simple cutoffs for these biomarkers identified individuals with good recovery for pain (positive predictive value [PPV], 0.85; 95% CI, 0.82-0.88), sleep (PPV, 0.63; 95% CI, 0.59-0.67, and anxiety (PPV, 0.76; 95% CI, 0.72-0.80) with high predictive value.Conclusions and RelevanceThese findings suggest that wrist-wearable device biomarkers may have utility as screening tools for pain, sleep, and anxiety symptom outcomes after trauma exposure in high-risk populations.

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Emotion dysregulation as a maintenance factor for posttraumatic stress symptoms: The role of anxiety sensitivity

et al. 2020

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Objectives The current study tested whether emotion dysregulation, assessed by the Difficulties in Emotion Regulation Scale (DERS), would predict posttraumatic stress symptoms (PTSS) through anxiety sensitivity (AS). Alternate cognitive‐affective mediators (i.e., distress tolerance and intolerance of uncertainty) were explored. Method A total of 259 trauma‐exposed community members (ages averaging 37.39; evenly distributed by gender) from a larger clinical trial targeting suicide risk factors completed a clinical interview and self‐report questionnaires at baseline, Week‐3, and 1‐month follow‐up appointments. Results AS at Week 3 significantly mediated the relationship between initial emotion dysregulation and 1‐month follow‐up posttraumatic stress symptoms (PTSS) after controlling for condition and baseline symptoms (β = .07; standard error = 0.03; 95% confidence interval = [0.01, 0.14]). Effects held for one out of six emotion dysregulation subscales. Distress tolerance and intolerance of uncertainty were not significant mediators (ps > .05). Conclusions This study demonstrates that emotion dysregulation may confer maintenance of PTSS through AS. Findings highlight potentially malleable targets for interventions.

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Anxiety Sensitivity Prospectively Predicts Posttraumatic Stress Symptoms Following a Campus Shooting

Cited by 55 publications

References 44 publications

Interactive Effects of Traumatic Brain Injury and Anxiety Sensitivity Cognitive Concerns on Post-traumatic Stress Among Active Duty Soldiers

Interactive Effects of Traumatic Brain Injury and Anxiety Sensitivity Cognitive Concerns on Post-traumatic Stress Among Active Duty Soldiers

Utility of Wrist-Wearable Data for Assessing Pain, Sleep, and Anxiety Outcomes After Traumatic Stress Exposure

Emotion dysregulation as a maintenance factor for posttraumatic stress symptoms: The role of anxiety sensitivity

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