Antimicrobial Susceptibility Patterns among Viridans Group Streptococcal Isolates from Infective Endocarditis Patients from 1971 to 1986 and 1994 to 2002

Prabhu, Rajesh M.; Piper, Kerryl E.; Baddour, Larry M.; Steckelberg, James M.; Wilson, Walter R.; Patel, Robin

doi:10.1128/aac.48.11.4463-4465.2004

Cited by 60 publications

(43 citation statements)

References 25 publications

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“…To date, however, the number of IE cases that have been reported as a result of VGS or S gallolyticus (bovis) strains that harbor any degree of penicillin resistance is small. [123][124][125][126] Therefore, it is difficult to define the optimal treatment strategies for this group of patients. Table 8 shows regimens for treatment of NVE caused by relatively penicillin-resistant strains (MIC >0.12-<0.5 µg/mL).…”

Section: Native Valvementioning

confidence: 99%

Infective Endocarditis in Adults: Diagnosis, Antimicrobial Therapy, and Management of Complications

Baddour¹,

Wilson²,

Bayer³

et al. 2015

Circulation

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2,450

1,421

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Background— Infective endocarditis is a potentially lethal disease that has undergone major changes in both host and pathogen. The epidemiology of infective endocarditis has become more complex with today’s myriad healthcare-associated factors that predispose to infection. Moreover, changes in pathogen prevalence, in particular a more common staphylococcal origin, have affected outcomes, which have not improved despite medical and surgical advances. Methods and Results— This statement updates the 2005 iteration, both of which were developed by the American Heart Association under the auspices of the Committee on Rheumatic Fever, Endocarditis, and Kawasaki Disease, Council on Cardiovascular Disease of the Young. It includes an evidence-based system for diagnostic and treatment recommendations used by the American College of Cardiology and the American Heart Association for treatment recommendations. Conclusions— Infective endocarditis is a complex disease, and patients with this disease generally require management by a team of physicians and allied health providers with a variety of areas of expertise. The recommendations provided in this document are intended to assist in the management of this uncommon but potentially deadly infection. The clinical variability and complexity in infective endocarditis, however, dictate that these recommendations be used to support and not supplant decisions in individual patient management.

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Section: Native Valvementioning

confidence: 99%

Infective Endocarditis in Adults: Diagnosis, Antimicrobial Therapy, and Management of Complications

Baddour¹,

Wilson²,

Bayer³

et al. 2015

Circulation

Self Cite

2,450

1,421

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“…Antibiotic resistance rates among viridans group streptococci have been increasing in the general community (Doern et al 1996 (Prabhu et al 2004). None of the strains of viridans streptococci were penicillin resistant in the earlier period whereas 13% of strains were resistant during the later period.…”

Section: Antibiotic Prophylaxis and Infective Endocarditismentioning

confidence: 99%

Bacteraemia due to dental flossing

Crasta

Daly

Mitchell

et al. 2009

J Clinic Periodontology

114

105

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Aims: The aims of this study were to (1) investigate the incidence of bacteraemia following flossing in subjects with chronic periodontitis or periodontal health; (2) identify the micro‐organisms in detected bacteraemias; and (3) identify any patient or clinical factors associated with such bacteraemia. Material and Methods: Baseline blood samples were obtained from 30 individuals with chronic periodontitis (17 M:13 F, 29–75 years) and 30 with periodontal health (17 M:13 F, 28–71 years) following a non‐invasive examination. Each subject's teeth were then flossed in a standardized manner and blood samples obtained 30 s and 10 min. after flossing cessation. Blood samples were cultured in a BACTEC™ system and positive samples subcultured for identification. Results: Forty per cent of periodontitis subjects and 41% of periodontally healthy subjects tested positive for bacteraemia following flossing. Viridans streptococci, which are commonly implicated in infective endocarditis (IE), were isolated from 19% of positive subjects and accounted for 35% of microbial isolates. Twenty per cent of subjects had a detectable bacteraemia at 10 min. post‐flossing. No patient or clinical factors were significantly associated with post‐flossing bacteraemia. Conclusions: Dental flossing can produce bacteraemia in periodontally healthy and periodontally diseased individuals at a rate comparable with that caused by some dental treatments for which antibiotic prophylaxis is given to prevent IE.

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“…Microbiologists and clinicians need a common lexicon to convey information about microorganisms that is biologically meaningful (10,11). Population-based genomic and proteomic approaches most likely are necessary to facilitate taxonomical classifications that encompass the microbial diversity of VGS from diverse environments and their unique features (i.e., virulence factors).…”

Section: Parasanguinis)mentioning

confidence: 99%

Phylogenetic Analysis of Viridans Group Streptococci Causing Endocarditis

et al. 2008

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Identification of viridans group streptococci (VGS) to the species level is difficult because VGS exchange genetic material. We performed multilocus DNA target sequencing to assess phylogenetic concordance of VGS for a well-defined clinical syndrome. The hierarchy of sequence data was often discordant, underscoring the importance of establishing biological relevance for finer phylogenetic distinctions.

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Antimicrobial Susceptibility Patterns among Viridans Group Streptococcal Isolates from Infective Endocarditis Patients from 1971 to 1986 and 1994 to 2002

Cited by 60 publications

References 25 publications

Infective Endocarditis in Adults: Diagnosis, Antimicrobial Therapy, and Management of Complications

Infective Endocarditis in Adults: Diagnosis, Antimicrobial Therapy, and Management of Complications

Bacteraemia due to dental flossing

Phylogenetic Analysis of Viridans Group Streptococci Causing Endocarditis

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