2006
DOI: 10.1590/s1413-86702006000100011
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Anti-glomerular basement membrane glomerulonephritis in an HIV positive patient: case report

Abstract: We report on a case of a patient with HIV infection, diagnosed 18 months prior to the development of an anti-glomerular basement membrane (anti-GBM) rapidly progressive glomerulonephritis; this is probably the first report of such an association. A 30-year-old white man presented with elevation of serum creatinine (1.3 -13.5 mg/dL within one month). At admission, the urinalysis showed proteinuria of 7.2 g/L and 8,000,000 erythrocytes/mL. Renal biopsy corresponded to a crescentic diffuse proliferative glomerulo… Show more

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Cited by 10 publications
(7 citation statements)
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“…The rarity of clinical cases of ANCA-associated vasculitis in HIV patients suggests that HIV may in fact lessen the chance of developing active vasculitis [26]. Similarly, there has been only 1 case report on anti-GBM disease in a patient with HIV [27]. The reasons why these vasculitides are rare in patients with HIV are unknown.…”
Section: Hiv-infected Patients With Kidney Disease Other Than Hivanmentioning
confidence: 99%
“…The rarity of clinical cases of ANCA-associated vasculitis in HIV patients suggests that HIV may in fact lessen the chance of developing active vasculitis [26]. Similarly, there has been only 1 case report on anti-GBM disease in a patient with HIV [27]. The reasons why these vasculitides are rare in patients with HIV are unknown.…”
Section: Hiv-infected Patients With Kidney Disease Other Than Hivanmentioning
confidence: 99%
“…We propose that stochastic Cellular Automata simulation is the most appropriate technique to investigate the therapeutic outcome of drug prescriptions in combination with interrupted treatment by plasma apheresis, which has been suggested in some reports to be a promising treatment option for patients in the later stages of HIV infection [20]- [26].…”
Section: Discussionmentioning
confidence: 99%
“…To our knowledge, only three cases of HIV infection and concomitant anti-GBM disease have been reported. [3][4][5] Each case had a different presentation, course of treatment, and clinical outcome ( Table 2). Antibody formation to the GBM has been linked to a conformational change in quaternary structure of the a-345 noncollagenous-1 (NC1) hexamer.…”
Section: Discussionmentioning
confidence: 99%