“…In the cat, the cervical vagus is contiguous with the cervical sympathetic trunk as the vagosympathetic trunk, which, closely bound to the common carotid artery, lies with the external jugular vein in the space between the longus capitus muscle and the trachea (Reighard and Jennings 1963). This conclusion is supported by similar observations of concomitant Homer's syndrome and laryngeal hemiplegia in the horse attributable to disruption of the cervical vagosympathetic trunk from various causes (Simoens andothers 1990, Green andothers 1992).…”
Horner's syndrome is described in three cats associated with wounds to the ventrolateral neck. In each case, ipsilateral laryngeal hemiplegia was observed on laryngoscopy. This finding provided strong evidence to support a diagnosis of second order Horner's syndrome due to disruption of the cervical sympathetic trunk, as motor fibres innervating laryngeal abductors also traverse the neck; both as descending fibres within the contiguous cervical vagus and as ascending fibres within the recurrent laryngeal nerve. Notably, the ability to vocalise was unimpaired in all cases and, in two cats, neck wounds were not apparent until the neck had been clipped and closely examined. These findings indicate that assessment of laryngeal function is of value when localising the site of the neural defect responsible for selected cases of second order Horner's syndrome.
“…In the cat, the cervical vagus is contiguous with the cervical sympathetic trunk as the vagosympathetic trunk, which, closely bound to the common carotid artery, lies with the external jugular vein in the space between the longus capitus muscle and the trachea (Reighard and Jennings 1963). This conclusion is supported by similar observations of concomitant Homer's syndrome and laryngeal hemiplegia in the horse attributable to disruption of the cervical vagosympathetic trunk from various causes (Simoens andothers 1990, Green andothers 1992).…”
Horner's syndrome is described in three cats associated with wounds to the ventrolateral neck. In each case, ipsilateral laryngeal hemiplegia was observed on laryngoscopy. This finding provided strong evidence to support a diagnosis of second order Horner's syndrome due to disruption of the cervical sympathetic trunk, as motor fibres innervating laryngeal abductors also traverse the neck; both as descending fibres within the contiguous cervical vagus and as ascending fibres within the recurrent laryngeal nerve. Notably, the ability to vocalise was unimpaired in all cases and, in two cats, neck wounds were not apparent until the neck had been clipped and closely examined. These findings indicate that assessment of laryngeal function is of value when localising the site of the neural defect responsible for selected cases of second order Horner's syndrome.
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