“…These include inhibition of L3 migration and L4 motility with Ascaris suum, as well as L1 feeding inhibition, adult motility assays of Ostertagia ostertagi and Cooperia oncophora, and larval exsheathment inhibition of Haemonchus contortus and Trichostrongylus colubriformis of L3 larvae (Brunet and Hoste 2006;Quijada et al, 2015;Desrues et al, 2016a;Ramsay et al, 2016). These in vivo trials found better host resilience and lower fecal egg counts of H. contortus, Teladorsagia circumcincta, and T. colubriformis and of a mixed gastrointestinal nematode infection, which were attributed to lower worm fecundity (Niezen et al, 1995(Niezen et al, , 1998aPaolini et al, 2005;Landau et al, 2010), and also lower Eimeria oocyst counts . These in vivo trials found better host resilience and lower fecal egg counts of H. contortus, Teladorsagia circumcincta, and T. colubriformis and of a mixed gastrointestinal nematode infection, which were attributed to lower worm fecundity (Niezen et al, 1995(Niezen et al, , 1998aPaolini et al, 2005;Landau et al, 2010), and also lower Eimeria oocyst counts .…”