Anorexia Nervosa �Feeding Disorder�, �Nervous Malnutrition� or �Weight Phobia�?

Ah, Crisp

doi:10.1159/000387594

Cited by 128 publications

(63 citation statements)

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“…Anorexia nervosa (AN) is an overall malnutritional state with multiple deficiencies of micronutrients which, anecdotally, displays an unexpected pattern of increased resistance to infections and malignancies (22)(23)(24)(25)(26)(27)(28)(29). Studies of the immune function of small groups of AN patients have revealed neutropenia with defective granulocyte bactericidal action and decreased adherence, normal or reduced chemotaxis, unchanged T lymphocyte subsets, normal numbers of B lymphocytes and normal serum immunoglobulin concentrations, increased humoral-antibody response to the influenza virus, highly variable or increased lymphocyte proliferative responses to mitogen stimulation, a relatively modest impairment in delayedhypersensitivity skin testing, normal or increased interferon-y production and plasma interleukin-1 concentrations, decreased serum complement proteins, normal plasma tumor necrosis factor (TNF) concentrations with in creased in vitro TNF spontaneous production by periph eral-blood mononuclear cells (PBMC) and normal TNF response to phytohemagglutinin (PHA), reduced cellmediated cytotoxicity, and elevated acute-phase reac tants.…”

Section: Introductionmentioning

confidence: 99%

Psychoimmunoendocrine Investigation in Anorexia nervosa

et al. 1993

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Immunological and neuroendocrine parameters were examined in 11 women with anorexia nervosa, 6 restricted and 5 bulimic-anorectics, 17-43 years old with 2-15 years duration of the disease, and in 11 age- and sex-matched psychophysically healthy controls. The T lymphocyte proliferative response to phytohemagglutinin (PHA), plasma adrenocorticotropic hormone (ACTH), cortisol and β-endorphin (β-EP) levels was examined in basal conditions and after corticotropin-releasing hormone (CRH) stimulation. Cortisol inhibition by dexamethasone (DST), and basal growth hormone (GH) and prolactin (PRL) levels were also examined. The immune study did not reveal significant differences between patients and controls. ACTH and cortisol basal levels were significantly higher in anorectics, while β-EP, GH and PRL concentrations did not differ in the two groups. ACTH, β-EP and cortisol responses to CRH were blunted in anorectics and the DST impaired in 55% of the patients. No correlations were observed between neuroendocrine impairments and the T lymphocyte response to PHA, or between the immunological neuroendocrine parameters and the body mass index of either patients or controls.

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Section: Introductionmentioning

confidence: 99%

Psychoimmunoendocrine Investigation in Anorexia nervosa

et al. 1993

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“…The sample was composed of all normal weight bulimic patients who had a sister or sisters, seen consecutively over a 40-month period. To enter the study all patients were diagnosed by the author as having bulimia according to the criteria of DSM-IIIR (American Psychiatric Association, 1985) but excluding patients with anorexia nervosa (Crisp, 1970) or massive obesity (Garrow, 1979).…”

Section: The Samplementioning

confidence: 99%

A comparative study of menarchal age and weight of bulimic patients and their sisters

Lacey

1992

Int. J. Eat. Disord.

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Sixty‐six consecutive bulimia nervosa patients and their sisters were compared for menar‐chal age and weight. Bulimic women were significantly heavier, but not younger, at puberty then their sisters being brought up in the same home environment. The belief that the bulimics were relatively overweight at menarche was held not only by the future patients but also by their sisters and mothers. At the very least the data represent a common family myth prior to the development of the illness. Interviews suggested that the issue of shape and weight became mutually overvalued and led to the initial aberrant eating behavior. Heavier menarchal weight appears to be one of the factors which prime a susceptible girl to future bulimia.

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“…Crisp (1970) suggested that homosexual conflict in the male might play a role comparable with that of the heterosexual threat in the female. Crisp (1983) maintains that there is usually profound gender uncertainty present at the outset, which may be either a recent pubertal phenomenon or chronically entrenched within an older adolescent.…”

Section: Homosexual Conflict?mentioning

confidence: 99%

Anorexia nervosa in the male: A review of clinical, epidemiological and biological findings

Scott¹

1986

Int. J. Eat. Disord.

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This paper collates previous reports of anorexia nervosa in males. Although as inclusive as possible, greater emphasis is given to certain papers in order to illustrate particular aspects of the male condition. Reasons are proposed as to why the diagnosis in males has been overlooked by many practitioners and theorists. The clinical picture and epidemiological features are described. The main endocrinological factors are presented and compared with those occurring in females. A sociocultural model is offered as one best integrating our knowledge of male anorexics. Running throughout is the general impression that anorexia nervosa within males is a counterpart to female anorexia. The applicability of the Feighner criteria to males is queried.

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Anorexia Nervosa �Feeding Disorder�, �Nervous Malnutrition� or �Weight Phobia�?

Cited by 128 publications

References 0 publications

Psychoimmunoendocrine Investigation in Anorexia nervosa

Psychoimmunoendocrine Investigation in Anorexia nervosa

A comparative study of menarchal age and weight of bulimic patients and their sisters

Anorexia nervosa in the male: A review of clinical, epidemiological and biological findings

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