Animal models of muscle diseases. Part II: Murine dystrophy

Cosmos, Ethel; Butler, Jane; Mazliah, Jacob; Allard, E. Paul

doi:10.1002/mus.880030413

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Cited by 7 publications

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“…This observation is in general agreement with the report by Cosmos, Butler, Mazliah & Allard (1980) on growth curves for the mouse mutant 129/ReJ-dy. However, pituitary gland weights were not consistently reduced except in the male and female CHF-147 hamsters.…”

Section: Resultssupporting

confidence: 93%

Abnormal gonadotrophin release from pituitaries of muscular dystrophic mice and hamsters

Wilkinson¹

1984

Reproduction

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Section: Resultssupporting

confidence: 93%

Abnormal gonadotrophin release from pituitaries of muscular dystrophic mice and hamsters

Wilkinson¹

1984

Reproduction

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Genetic regulation of slowly progressing mild muscle atrophy in fast-twitch muscles of BUF/Mna rats

et al. 1997

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Regeneration and reinnervation of the dystrophic mouse soleus muscle

Summers

Ashmore

1983

Acta Neuropathol

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The regeneration and reinnervation of the dystrophic mouse soleus muscle was investigated in response to a double crush-lesion, which causes degeneration of muscle fibres leaving the innervation intact. In normal and dystrophic muscles, injury produced degeneration of muscle fibres, proliferation and fusion of muscle satellite cells, and growth and reinnervation of regenerating fibres. Four, 6 and 21 days after injury, regenerating dystrophic fibres were 50% smaller in cross-sectional area than regenerating normal fibres and showed several pathological changes. Nerve terminal morphology was initially unaffected by the crush, and nerve terminals were associated with degenerating muscle fibres 2 days after injury and with regenerating muscle fibres 6-28 days after crushing. In intact muscles dystrophic endplates were longer and showed increased ultraterminal sprouting compared to normal endplates. At 28 days after crushing normal nerve terminal sprouting was significantly increased compared to the contralateral control. The extent of nerve terminal sprouting and endplate length in dystrophic muscles was not affected by the degeneration and subsequent regeneration of the muscle fibres. We conclude that a proportion of dystrophic mouse soleus muscle fibres can regenerate after a crush when the innervation is left intact.

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Animal models of muscle diseases. Part II: Murine dystrophy

Cited by 7 publications

References 0 publications

Abnormal gonadotrophin release from pituitaries of muscular dystrophic mice and hamsters

Abnormal gonadotrophin release from pituitaries of muscular dystrophic mice and hamsters

Genetic regulation of slowly progressing mild muscle atrophy in fast-twitch muscles of BUF/Mna rats

Regeneration and reinnervation of the dystrophic mouse soleus muscle

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