Angiosarcoma of the Thigh with Thrombosis in the Femoral Artery

Miyamoto, T.; Mihara, Motoyuki; Iwasaki, Kazumi; Mishima, Erika; Shimao, Shuhei; Miyamoto, Hiroaki

doi:10.1111/j.1346-8138.1992.tb03216.x

Cited by 6 publications

(4 citation statements)

References 6 publications

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“…It is instructive to explore whether in the general case given by Eq. ( 12), which can be denoted as modified Jacobi ellipsoids following Miyamoto (1967), the same qualitative behaviour can be observed, as in the case of pure Jacobi ellipsoids. Figure 4 illustrates the scenario for a particular case with a given set of geometrical and kinematic parameters.…”

Section: The Case For Deformable Rotationmentioning

confidence: 59%

Dynamical evolution of two associated galactic bars

Garzón

López-Corredoira

2014

Astron Nachr

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We study the dynamical interactions of mass systems in equilibrium under their own gravity that mutually exert and experience gravitational forces. The method we employ is to model the dynamical evolution of two isolated bars, hosted within the same galactic system, under their mutual gravitational interaction. In this study we present an analytical treatment of the secular evolution of two bars that oscillate with respect one another. Two cases of interaction, with and without geometrical deformation, are discussed. In the latter case, the bars are described as modified Jacobi ellipsoids. These triaxial systems are formed by a rotating fluid mass in gravitational equilibrium with its own rotational velocity and the gravitational field of the other bar. The governing equation for the variation of their relative angular separation is then numerically integrated, which also provides the time evolution of the geometrical parameters of the bodies. The case of rigid, non-deformable, bars produces in some cases an oscillatory motion in the bodies similar to that of a harmonic oscillator. For the other case, a deformable rotating body that can be represented by a modified Jacobi ellipsoid under the influence of an exterior massive body will change its rotational velocity to escape from the attracting body, just as if the gravitational torque exerted by the exterior body were of opposite sign. Instead, the exchange of angular momentum will cause the Jacobian body to modify its geometry by enlarging its long axis, located in the plane of rotation, thus decreasing its axial ratios.Comment: Accepted for publication in A

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Section: The Case For Deformable Rotationmentioning

confidence: 59%

Dynamical evolution of two associated galactic bars

Garzón

López-Corredoira

2014

Astron Nachr

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“…8 Thus, clinical symptoms usually relate to the level of aortic lumen obstruction and the presence or absence of thromboembolic metastasis; in addition, preoperative diagnosis is difficult because intimal angiosarcomas present with diverse and nonspecific symptoms, and imaging studies often fail to differentiate among atheroma, mural thrombus, aneurysm, and tumors. [2][3][4]9,10 According to the histologic site of origin, primary malignant tumors of arteries have been classified as mural tumors when they originate from the media or adventitia (these tumors usually present as aneurysms or masses), and luminal tumors originate from the intima, forming a polypoid mass that grows along the lumen of the vessel, presenting as luminal obstructions and/or embolic events. 6 However, published cases of intimal angiosarcoma presenting with aneurysm and/or arterial rupture are very rare (15 cases: 14 involving the aorta and one involving the carotid artery), and preoperative and intraoperative clinical and radiological evidence could not predict a malignant condition.…”

Section: Discussionmentioning

confidence: 99%

“…1 Among them, exceptionally rare cases of intimal angiosarcoma have been reported to arise from large vessels such as the aorta or common femoral artery (CFA) or superficial femoral artery (SFA), presenting with aortic occlusion or lower extremity arterial stenosis or embolization. [2][3][4][5] Here, we report a patient who initially presented with a CFA aneurysm.…”

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confidence: 93%

Intimal angiosarcoma presenting with common femoral artery aneurysm

Choi

Min

Kim

et al. 2012

Journal of Vascular Surgery

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“…Vascular tumors may sometimes be related to the pathogenesis of thrombosis in other sites due to endothelial damage, although the mechanisms are not clear (Miyamoto et al, 1992). Multiple hemorrhagic and thrombotic episodes involving vascular tumors can lead to thrombocytopenic and hemorrhagic crises (Herman et al, 2002).…”

Section: Other Chemicals That Induced Atrial Thrombosis Related To Vamentioning

confidence: 99%

Chemical-Induced Atrial Thrombosis in NTP Rodent Studies

Yoshizawa

Kissling

Johnson³

et al. 2005

Toxicol Pathol

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Cardiac thrombosis, one of the causes of sudden death throughout the world, plays a principal role in several cardiovascular diseases, such as myocardial infarction and stroke in humans. Data from studies of induction of chemical thrombosis in rodents help to identify substances in our environment that may contribute to cardiac thrombosis. Results for more than 500 chemicals tested in rodents in 2-year bioassays have been published as Technical Reports of the National Toxicology Program (NTP) http://ntp-server.niehs.nih.gov/index . We evaluated atrial thrombosis induced by these chemical exposures and compared it to similarly induced lesions reported in the literature. Spontaneous rates of cardiac thrombosis were determined for control Fischer 344 rats and B6C3F1 mice: 0% in rats and mice in 90-day studies and, in 2-year studies, 0.7% in both genders of mice, 4% in male rats, and 1% in female rats. Incidences of atrial thrombosis were increased in high-dosed groups involving 13 compounds (incidence rate: 20-100%): 2-butoxyethanol, C.I. Direct Blue 15, bis(2-chloroethoxy)methane, diazoaminobenzene, diethanolamine, 3,3 -dimethoxybenzidine dihydrochloride, hexachloroethane, isobutene, methyleugenol, oxazepam, C.I. Pigment Red 23, C.I. Acid Red 114, and 4,4 -thiobis(6-t-butyl-m-cresol). The main localization of spontaneously occurring and chemically induced thromboses occurred in the left atrium. The literature survey suggested that chemicalinduced atrial thrombosis might be closely related to myocardial injury, endothelial injury, circulatory stasis, hypercoagulability, and impaired atrial mechanical activity, such as atrial fibrillation, which could cause stasis of blood within the left atrial appendage, contributing to left atrial thrombosis.Supplementary data referenced in this paper are not printed in this issue of Toxicologic Pathology. They are available as downloadable files at http:taylorandfrancis.metapress.com/openurl.asp?genre=journal&issn=0192-6233. To access them, click on the issue link for 33(5), then select this article. A download option appears at the bottom of this abstract. In order to access the full article online, you must either have an individual subscription or a member subscription accessed through www.toxpath.org.

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Angiosarcoma of the Thigh with Thrombosis in the Femoral Artery

Cited by 6 publications

References 6 publications

Dynamical evolution of two associated galactic bars

Dynamical evolution of two associated galactic bars

Intimal angiosarcoma presenting with common femoral artery aneurysm

Chemical-Induced Atrial Thrombosis in NTP Rodent Studies

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