Anatomy; histology, and clinical dysfunction of the cricopharyngeus muscle

Shapiro, Jo; Kelly, James H.

doi:10.1097/00020840-199402000-00011

Cited by 4 publications

(3 citation statements)

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“…Demographic and dysphagia symptom data for the 12 participants are included in Tables 1 and 2. Etiologies varied within the group and included peripheral neuropathy (5), laryngectomy/pharyngectomy (3), isolated CPM spasm (2), and CVA (2).…”

Section: Subjectsmentioning

confidence: 99%

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Botulinum Toxin Treatment for Cricopharyngeal Dysfunction

Shaw

Searl

2001

Dysphagia

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Hypertonicity and spasticity of the cricopharyngeal muscle (CPM) often result in dysphagia characterized by difficulty passing a bolus through the upper esophageal sphincter. Past treatments for this problem have included mechanical dilation and endoscopic and transcervical cricopharyngeal myotomy. More recently, botulinum toxin injections into the CPM have been successful, but only in isolated case studies and small series. This study reports pre- and post-botulinum toxin A injection results for 12 subjects, including patient ratings of symptom severity, changes noted during modified barium swallow studies, and, in some cases, manometry of the upper esophageal sphincter. Results indicate that botulinum toxin A treatment provided significant improvement in swallowing as indicated by patient symptom ratings and investigator ratings of function from modified barium swallow studies. Greater improvement was seen in those with more isolated CPM or Xth nerve dysfunction rather than those with more global dysphagia abnormalities.

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Section: Subjectsmentioning

confidence: 99%

“…It is classically described as consisting of the lower oblique ®bers of the inferior constrictor muscle, the cricopharyngeus (CPM), and the upper circular ®bers of the esophagus [2]. This sphincteric structure separates the hypopharynx from the esophagus.…”

mentioning

confidence: 99%

Botulinum Toxin Treatment for Cricopharyngeal Dysfunction

Shaw

Searl

2001

Dysphagia

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“…However, UES opening was not obtained irrespective of the elevation of the hyoid bone and the larynx in this lateral medullary syndrome patient. The UES consists of the cricopharyngeus muscle, the lower fibers of the inferior pharyngeal constrictor, and the upper circular fibers of the esophagus; however, the cricopharyngeus muscle is the main component (18). The cricopharyngeus muscle usually contracts and then relaxes to pass a bolus.…”

Section: Fes In a Lateral Medullary Syndrome Patientmentioning

confidence: 99%