Anatomy and Embryology of Cerebral Circulation

Bergh, R. Van den; Eecken, Henri Van Der

doi:10.1016/s0079-6123(08)61433-8

Cited by 73 publications

(28 citation statements)

References 48 publications

Supporting

Mentioning

Contrasting

Unclassified

Order By: Relevance

“…All confirmed the presence of LMA. [62][63][64] Moreover, Wollschlaeger and Wollschlaeger, 65 who successfully injected 58 human cadaver brains, observed LMA, as described by Vander Eecken. 14, 15 Lazorthes et al 59 were some of the last anatomists to publish findings on LMA.…”

Section: Section 1: Anatomic Evidencementioning

confidence: 89%

Anatomy and Functionality of Leptomeningeal Anastomoses

Brozici

Zwan

Hillen

2003

Stroke

219

200

View full text Add to dashboard Cite

Background-This review seeks to provide a structured presentation of existing knowledge of leptomeningeal anastomoses from anatomic and functional points of view and to identify problems and possible research directions to foster a better understanding of the subject and of stroke mechanisms. Summary of Review-Available data show that leptomeningeal anastomoses may be important in understanding stroke mechanisms and that leptomeningeal anastomoses play an important role in penumbra outcome. However, the literature shows no consensus between statements on the existence of leptomeningeal anastomoses and compensatory capacity. Conclusions-By

show abstract

Section: Section 1: Anatomic Evidencementioning

confidence: 89%

Anatomy and Functionality of Leptomeningeal Anastomoses

Brozici

Zwan

Hillen

2003

Stroke

219

200

View full text Add to dashboard Cite

show abstract

“…[23][24][25] In contrast, the subcortical white matter is not an arterial watershed area. 26 Atherosclerosis induces hyalinization, tortuosity, and elongation of vessels in the periventricular white matter. 24,[27][28][29] This may contribute to a decrease in blood flow in the periventricular white matter, leading to ischemia.…”

Section: Discussionmentioning

confidence: 99%

Aortic Atherosclerosis at Middle Age Predicts Cerebral White Matter Lesions in the Elderly

Leeuw

Groot

Oudkerk

et al. 2000

Stroke

104

View full text Add to dashboard Cite

Background and Purpose-MRI scans of the brains of elderly people frequently show white matter lesions. Clinically, these lesions are associated with cognitive impairment and dementia. A relation between atherosclerosis and white matter lesions was found in some small cross-sectional studies. However, atherosclerosis is a gradual process that starts early in life. We investigated the longitudinal association between aortic atherosclerosis assessed during midlife and late life and cerebral white matter lesions. Methods-We randomly sampled subjects between 60 and 90 years old from 2 population-based follow-up studies in which subjects had their baseline examinations in 1975 to 1978 (midlife) and in 1990 to 1993 (late life). In 1995 to 1996, subjects underwent 1.5-T MRI scanning; white matter lesions were rated in the deep subcortical and periventricular regions separately. Aortic atherosclerosis was assessed on abdominal radiographs that were obtained from 276 subjects in midlife and 531 subjects in late life. Results-The presence of aortic atherosclerosis during midlife was significantly associated with the presence of periventricular white matter lesions Ϸ20 years later (adjusted relative risk, 2.4; 95% CI, 1.2 to 5.0); the relative risks increased linearly with the severity of aortic atherosclerosis. No association was found between midlife aortic atherosclerosis and subcortical white matter lesions (adjusted relative risk, 1.1; 95% CI, 0.5 to 2.3) or between late-life aortic atherosclerosis and white matter lesions. Conclusions-The pathogenetic process that leads to cerebral periventricular white matter lesions starts already in or before midlife. The critical period for intervention directed at prevention of white matter lesions and its cognitive consequences may be long before these lesions become clinically detectable. (Stroke. 2000;31:425-429.)

show abstract

“…The lesion was first described by Virchow in 1867; and was reemphasized and named periventricular leucomalacia by Banker and Larroche (1962) who described 51 cases and reviewed the published cases. This unusual site for cerebral infarction occurs in the watershed area between the ventriculopetal and ventriculofugal circulations described by Van den Bergh and Vander Eecken (1968). In neonates the infarction results from failure of perfusion (ischaemia).…”

mentioning

confidence: 97%

Periventricular leucomalacia in neonates: Complications and sequelae

Armstrong¹,

Norman²

1974

Archives of Disease in Childhood

166

View full text Add to dashboard Cite

. Periventricular leucomalacia in neonates: complications and sequelae. Periventricular leucomalacia was examined in a total of 28 infants. These foci of infarction were attributed to episodes of failure of perfusion due to hypotension in a border zone between ventriculofugal and ventriculopetal circulations. We describe haemorrhage occurring into these infarctions as a complication occurring in 7 infants. In 2 infants with a bleeding diathesis the haemorrhage was massive and fatal.

show abstract

Anatomy and Embryology of Cerebral Circulation

Cited by 73 publications

References 48 publications

Anatomy and Functionality of Leptomeningeal Anastomoses

Anatomy and Functionality of Leptomeningeal Anastomoses

Aortic Atherosclerosis at Middle Age Predicts Cerebral White Matter Lesions in the Elderly

Periventricular leucomalacia in neonates: Complications and sequelae

Contact Info

Product

Resources

About