Analysis of sweat evaporation from clothing materials by the ventilated sweat capsule method

Otomasu, Kinuyo; Yamauchi, Masaki; Ohwatari, Nobu; Matsumoto, Takaaki; Tsuchiya, Katsuhiko; Kosaka, Mitsuo

doi:10.1007/s004210050205

Cited by 7 publications

(5 citation statements)

References 18 publications

(13 reference statements)

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“…When a heat stimulus is applied without exercise, the main cardiovascular stress is heat dissipation, of which sweating is a major component. However, once exercise is added or substituted, the main cardiovascular stress becomes the exercise itself, and sweating becomes a relatively minor portion of the total cardiovascular load [28][29][30][31][32][33].…”

Section: Discussionmentioning

confidence: 99%

The clinical thermoregulatory sweat test induces maximal seating

Hsieh

McNeeley

Chelimsky

2001

Clinical Autonomic Research

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Although thermoregulatory sweat testing is commonly used to assess the autonomic nervous system, the power of this stimulus to induce sweating has not been studied. In 8 healthy male subjects, the authors quantitated sweat rates, core temperature, heart rate, and blood pressure during clinical thermoregulatory sweat testing, a separate exercise protocol, and with exercise added to thermal conditions. The authors found that (1) the addition of exercise to the thermal environment produced no further increase in sweat rate (3,841+/-948 versus 3,888+/-866 nl/mn - cm2); (2) maximum sweat rates closely corresponded to the theoretical maximum (6,000 nl/mn - cm2) derived from single gland studies; (3) sweat rates vary across subjects, but are similar across sites in any one individual; (4) core temperature rise is a major determinant of cardiovascular load in both thermal and exercise settings; (5) blood pressure decreased 28/11 mm Hg during thermal load, but increased 26/10 mm Hg with exercise, in agreement with current understanding of muscle and skin vascular physiology. The authors conclude that clinical thermoregulatory testing conditions produce maximum sweat rates in humans.

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Section: Discussionmentioning

confidence: 99%

The clinical thermoregulatory sweat test induces maximal seating

Hsieh

McNeeley

Chelimsky

2001

Clinical Autonomic Research

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“…They rested for 30 min and then immersed their leg in water at 42°C for 30 min. Immersion of the leg in hot water has been established as a standard method of enhancing sweating and raising body temperature, and been used experimentally to investigate effects of heating subjects (Matsumoto et al 1993;Otomasu et al 1997).…”

Section: Test Immersion Exposuresmentioning

confidence: 99%

No effects of acclimation to heat on immune and hormonal responses to passive heating in healthy volunteers

et al. 2011

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Heat acclimation results in whole body-adaptations that increase heat tolerance, and might also result in changed immune responses. We hypothesized that, after heat acclimation, tumor necrosis factor alpha, interleukin 6 and the lymphocyte count would be altered. Heat acclimation was induced in 6 healthy men by 100 min of heat exposure for 9 days. Heat exposure consisted of (1) 10 min of immersion up to chest-level in water at 42°C and (2) 90 min of passive heating by a warm blanket to maintain tympanic temperature at 37.5°C. The climatic chamber was maintained at 40°C and a relative humidity of 50%. Blood samples were analyzed before and after heat acclimation for natural killer (NK) cell activity, counts of lymphocytes B and T, before and after heat acclimation for peripheral blood morphology, interleukin 6, tumor necrosis factor alpha, and cortisol. A Japanese version of the profile of mood states questionnaire was also administered before and after acclimation. The concentrations of white blood cells, lymphocytes B and T, cortisol, interleukin 6, tumor necrosis factor alpha and NK cell activity showed no significant differences between pre- and post-acclimation, but there was a significantly lower platelet count after acclimation and, with the profile of mood states questionnaire, there was a significant rise in anger after acclimation. It is concluded that heat acclimation by passive heating does not induce alterations in immune or endocrine responses.

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“…The subjects rested for 30 min and then immersed their leg in water at 42°C for 30 min. Immersion of the leg in hot water has been established as a standard method to enhance sweating and body temperature (Matsumoto et al 1993;Otomasu et al 1997). This intervention was performed once in the afternoon.…”

Section: Subjects and Protocolmentioning

confidence: 99%

Influence of season on plasma antidiuretic hormone, angiotensin II, aldosterone and plasma renin activity in young volunteers

et al. 2009

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We investigated seasonal changes in hormonal and thermoregulatory responses. Eight volunteers were subjected to the experiment at four times of the year: around the vernal and autumnal equinoxes, and at the summer and winter solstices at latitude 35 degrees N. Plasma antidiuretic hormone (ADH), angiotensin II (ANG II), aldosterone (ALD) and plasma renin activity (PRA) were analyzed before and after water immersion. Seasonal changes in thermoregulatory responses were assessed by measuring core temperature and sweat rate during immersion of the leg in hot water (at 42 degrees C) for 30 min in a room maintained at 26 degrees C. The concentration of plasma ADH and ALD before water immersion was significantly higher in summer than in other seasons. The concentrations of ANG II and PRA did not show seasonal variations. Changes in tympanic temperature during water immersion showed significant differences between seasons, and were higher in winter than in other seasons. The sweat rate was significantly higher in summer than in other seasons. In summary, ADH and ALD concentrations displayed a seasonal rhythm with marked elevation in summer; this may be a compensative mechanism to prevent dehydration from increased sweat loss during summer due to heat acclimatization.

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Analysis of sweat evaporation from clothing materials by the ventilated sweat capsule method

Cited by 7 publications

References 18 publications

The clinical thermoregulatory sweat test induces maximal seating

The clinical thermoregulatory sweat test induces maximal seating

No effects of acclimation to heat on immune and hormonal responses to passive heating in healthy volunteers

Influence of season on plasma antidiuretic hormone, angiotensin II, aldosterone and plasma renin activity in young volunteers

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