“…As a consequence, further chloroplast development does not proceed. Transcripts encoding plastid proteins, such as the small subunit of ribulose-I ,5-bisphosphate carboxylase and the light-harvesting chlorophyll-db-binding proteins, as well as mRNAs for peroxisomal and other proteins related to plastid function rapidly disappear (Reiss et al, 1983;Mayfield and Taylor, 1984;Harpster et al, 1984;Oelmiiller and Mohr, 1986;Oelmuller et al, 1986aOelmuller et al, , 1988Oelmuller and Schuster, 1987) because their nuclear genes are no longer transcribed (Simpson et al, 1986;Stockhaus et al, 1987;Ernst and Schefieck, 1988;Hess et al, 1994). Similar to nortlurazon, nuclear mutations that cause carotenoid deficiency and thus ultimately lead to photooxidation of the plastid compartment negatively influence the expression of the nuclear rbcS and Zhcdb genes (Mayfield and Taylor, 1984;Batschauer et al, 1986;Burgess and Taylor, 1987).…”