2018
DOI: 10.1016/j.bpj.2017.11.2381
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Amyloid-β Peptide Interaction with Lipid Bilayer Promotes Peptide Aggregation on the Surface and Modulates Lipid Behavior

Abstract: Abnormal (misfolded) form of human prion protein displays high propensity towards self-association which may result in formation of insoluble fibrillar

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