Abstract:Exercise can improve obesity and metabolic disorders in mice fed a high-fat diet (HFD), but the role of AMPKa2 in the process remains unclear. The aim of this study was to investigate the role of AMPKa2 in the exercise-induced improvements in glucose tolerance and metabolic turnover in obesity mice. Male wild-type mice (n512) and AMPKa2 knockout (AMPKa2 KO) mice (n512) were fed a HFD for 16 wk and were then randomly divided into four groups: WT HFD group (WT HF), AMPKa2 KO HFD group (AMPKa2 KO HF), WT HFD exer… Show more
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