Ambulatory blood pressure monitoring in renal transplant patients: modifiable parameters after active antihypertensive treatment

Oliveras, Anna; Vázquez, Susana; Hurtado, Sara; Vila, Jordi; Puig, Josep; Lloveras, J

doi:10.1016/j.transproceed.2004.04.085

Cited by 8 publications

(4 citation statements)

References 19 publications

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“…The prevalence of post-transplant hypertension reported in the literature varies considerably depending on the study population and the criteria used to define hypertension [3,6]. In adults, the incidence of post-transplant hypertension has been estimated between 60% and 80% [7]. In one cross-sectional study of 409 adult subjects with stable kidney allograft function, the prevalence of hypertension (defined as BP > 150/90 mm Hg) was 77.3% [8].…”

Section: Incidence Prevalence and Characterization Of Post-transplamentioning

confidence: 99%

“…Post-transplant hypertension demonstrates a distinctive characteristic on ambulatory BP monitoring: a high prevalence of nocturnal hypertension [7]. In one prospective study, nearly 75% of subjects with posttransplant hypertension demonstrated absence or reversal of the normal nocturnal fall in BP (ie, nondippers) [7].…”

Section: Incidence Prevalence and Characterization Of Post-transplamentioning

confidence: 99%

Section: Incidence Prevalence and Characterization Of Post-transplamentioning

confidence: 99%

“…In one prospective study, nearly 75% of subjects with posttransplant hypertension demonstrated absence or reversal of the normal nocturnal fall in BP (ie, nondippers) [7]. Additionally, cyclosporine has been implicated as contributing to the loss of the normal nocturnal drop in BP [7]. In non-kidney transplant recipients, loss of nocturnal dip is associated with left ventricular hypertrophy, lacunar stroke, and microalbuminuria [9].…”

Section: Incidence Prevalence and Characterization Of Post-transplamentioning

confidence: 99%

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Is RAS blockade routinely indicated in hypertensive kidney transplant patients?

Salzberg

2007

Current Science Inc

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There is a high incidence of hypertension after kidney transplantation, which has been associated with the development of left ventricular hypertrophy, an increased risk for acute rejection, lower graft survival, and increased mortality. The pathogenesis of post-transplant hypertension is multifactorial, and therefore optimum therapy for it is not clearly defined. Historically, use of renin-angiotensin system (RAS) blockade in post-transplant hypertension has been limited given concerns of inducing worsening allograft function. Recent data demonstrated that subjects with post-transplant hypertension can be treated effectively with RAS blockers, and that these agents may offer significant additional benefits beyond blood pressure control. Review of the literature suggests that RAS blockers should be considered as useful agents for treatment of post-transplant hypertension not due to transplant renal artery stenosis.

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Section: Incidence Prevalence and Characterization Of Post-transplamentioning

confidence: 99%

Section: Incidence Prevalence and Characterization Of Post-transplamentioning

confidence: 99%