Amaurosis secundaria a traumatismo frontal

Santos-Bueso, E.; Genol-Saavedra, I; Calvo-González, C; Huelga-Zapico, E; Díaz-Valle, D.; Benítez-del-Castillo, J.M.; García–Sánchez, J.

doi:10.4321/s0365-66912006000200012

Cited by 2 publications

(2 citation statements)

References 3 publications

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“…This stress wave resulted in an uneven deformation of the upper and lower rim of the optic canal that would cause a predominantly shearing type injury in the optic nerve. Notably, Santos-Bueso et al (53) have reported a clinical case of ITON in which a patient suffered a right frontal impact, which is very similar with the 45 • case in our simulation.…”

Section: Discussionsupporting

confidence: 84%

The Biomechanics of Indirect Traumatic Optic Neuropathy Using a Computational Head Model With a Biofidelic Orbit

Singman

McCulley

et al. 2020

Front. Neurol.

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Indirect traumatic optic neuropathy (ITON) is an injury to the optic nerve due to head trauma and usually results in partial or complete loss of vision. In order to advance a mechanistic understanding of the injury to the optic nerve, we developed a head model with a biofidelic orbit. Head impacts were simulated under controlled conditions of impactor velocity. The locations of impact were varied to include frontal, lateral, and posterior parts of the head. Impact studies were conducted using two types of impactors that differed in their rigidity relative to the skull. The simulated results from both the impactors suggest that forehead impacts are those to which the optic nerve is most vulnerable. The mode and location of optic nerve injury is significantly different between the impacting conditions. Simulated results using a relatively rigid impactor (metal cylinder) suggest optic nerve injury initiates at the location of the intracranial end of the optic canal and spreads to the regions of the optic nerve in the vicinity of the optic canal. In this case, the deformation of the skull at the optic canal, resulting in deformation of the optic nerve, was the primary mode of injury. On the other hand, simulated results using a relatively compliant impactor (soccer ball) suggest that primary mode of injury comes from the brain tugging upon the optic nerve (from where it is affixed to the intracranial end of the optic canal) during coup countercoup motion of the brain. This study represents the first published effort to employ a biofidelic simulation of the full length of the optic nerve in which the orbit is integrated within the whole head.

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Section: Discussionsupporting

confidence: 84%

The Biomechanics of Indirect Traumatic Optic Neuropathy Using a Computational Head Model With a Biofidelic Orbit

Singman

McCulley

et al. 2020

Front. Neurol.

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show abstract

“…Regarding aetiology of traumatic optic neuropathy as the cause of vision impairment, different explanations have been stated, ranging from concussion and contusion along the orbital ceiling and optic canal to optic nerve shearing, haemorrhage, nerve sheath haematoma to oedema within the optic canal and chiasma or cortical insults 2–4 6 7 10 11. CT or MRI scans reveal a continuum of affections ranging from small lacerations and bleeding5 12–14 to major comminuted anterior skull base fractures 12 15 16. By now the exact pathogenesis of how a frontal impact causes changes within the optic canal remains unclear in patients, in which fracture lines crossing the optic nerve, optic canal compression or massive haematoma are absent 3 4…”

Section: Introductionmentioning

confidence: 99%