Alkaline reflux gastritis

Berardi, Romeo S.; D, Siroospour; R, Ruiz; Carnes, William H.; Devaiah, K.A.; Peterson, Carl A.; Becknell, William E.; Olivencia, Jose Antonio

doi:10.1016/0002-9610(76)90339-1

Cited by 19 publications

(3 citation statements)

References 15 publications

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“…Bile reflux is an important factor in the pathogenesis of gastritis and esophagitis following previous gastric operations [1][2][3][4]. Operations that aim to preserve the pylorus minimize the extent of duodenogastric reflux [5] and the highest incidence of bile reflux is reported after partial gastrectomy [6].…”

mentioning

confidence: 99%

Exposure to bile acids and bacteria over 24 hours following partial gastrectomy, vagotomy, and pyloroplasty

et al. 1986

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Hourly measurements of bile acids, bacterial flora, and pH were made from gastric aspirates collected over 24 hours in subjects taking a controlled diet. Significantly higher concentrations of total bile acids were found following truncal vagotomy and antrectomy (TV+A), 2.73±1.16 SEM mmol/l, than after truncal vagotomy and pyloroplasty (TV+P), 0.88±0.56 mmol/l (p<0.002), and controls, 0.43±0.22 mmol/l (p<0.002). These observations were due to differences in nocturnal reflux only. Free secondary bile acids were identified more frequently after TV+A (19% of aspirates) than TV+P (6%) or controls (2%).Streptococcus faecalis andVeillonella spp, the principal organisms known to deconjugate and hydrolyze bile acids, were present in 65% of aspirates after TV+A compared with 35% after TV+P, and 33% of the controls. Mean pH values were 5.12 after TV+A, 2.82 after TV+P, and 2.14 in the controls. We conclude that antrectomy is associated with a greater incidence of duodenal reflux and a more suitable environment for the formation of secondary bile acids than vagotomy and pyloroplasty or in normal subjects.

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mentioning

confidence: 99%

Exposure to bile acids and bacteria over 24 hours following partial gastrectomy, vagotomy, and pyloroplasty

et al. 1986

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show abstract

“…Παρόλα αυτά, ιστολογικά ευρήματα είναι συχνά σε ασθενείς χωρίς συμπτώματα. Έχει παρατηρηθεί μια φτωχή συσχέτιση μεταξύ των ιστολογικών ευρημάτων και της κλινικής εικόνας [66][67] .…”

Section: κεφαλαιο III α αξιολόγηση της κλινικής και ενδοσκοπικής εικόνας στην αλκαλική γαστρίτιδαunclassified

“…Σε μια μελέτη 67 Μια ποικιλία συμπτωμάτων μπορεί να οδηγήσουν σε ενδο σκόπηση ασθενείς οι οποίοι προηγουμένως είχαν εγχείρηση στομάχου. Όλα αυτά έχουν χαρακτηριστεί σαν το σύνδρομο της αλκαλικής γαστρίτιδας εκ παλινδρομήσεως 69 .…”

Section: κεφαλαιο III α αξιολόγηση της κλινικής και ενδοσκοπικής εικόνας στην αλκαλική γαστρίτιδαunclassified